Expression of miR-155 in infarcted hearts (day 3) of <i>db/db</i> mice receiving BMPC intramyocardial. by Raj Kishore (49181)

Knowledge and personal attitude scales by site. by Allison L. Scott (15927323)

Association between statins’ exposure with incidence and prognosis of gastric cancer: an updated meta‐analysis by Dandi Lou (13181970)

Data for "Irreversible Southern Ocean Silicate Front contraction and global nutrient depletion" by Dong-Geon Lee (17274778)

Expression of is down-regulated in high-risk adenomas and colorectal carcinoma-0 by Anders Lorentzen (79814)

Image_8_Cannabinoid WIN55,212-2 reprograms monocytes and macrophages to inhibit LPS-induced inflammation.tif by Mario Pérez-Diego (14799367)

“…Mechanistically, WIN55,212-2 induced a metabolic and epigenetic shift in macrophages by decreasing LPS-induced mTORC1 signaling, commitment to glycolysis and active histone marks in pro-inflammatory cytokine promoters. …”

Data_Sheet_1_Impact of Citrus Pulp or Inulin on Intestinal Microbiota and Metabolites, Barrier, and Immune Function of Weaned Piglets.pdf by Julie Uerlings (9035627)

“…Inulin (IN), a well-known prebiotic, was used for comparison. …”

Image_4_Cannabinoid WIN55,212-2 reprograms monocytes and macrophages to inhibit LPS-induced inflammation.tif by Mario Pérez-Diego (14799367)

“…Mechanistically, WIN55,212-2 induced a metabolic and epigenetic shift in macrophages by decreasing LPS-induced mTORC1 signaling, commitment to glycolysis and active histone marks in pro-inflammatory cytokine promoters. …”

Image_2_Cannabinoid WIN55,212-2 reprograms monocytes and macrophages to inhibit LPS-induced inflammation.tif by Mario Pérez-Diego (14799367)

“…Mechanistically, WIN55,212-2 induced a metabolic and epigenetic shift in macrophages by decreasing LPS-induced mTORC1 signaling, commitment to glycolysis and active histone marks in pro-inflammatory cytokine promoters. …”

Table_1_Identifying Age Based Maturation in the ERP Response to Faces in Children With Autism: Implications for Developing Biomarkers for Use in Clinical Trials.docx by Sara Jane Webb (8421699)

“…Our data demonstrate that the N170L and P100L responses to upright faces appeared to follow a roughly linear relationship with age. In the ASD group, the distribution of the age-adjusted residual values suggest that ASD participants were more likely to demonstrate slower latencies than would be expected for a TD child of the same age, similar to what has been identified using unadjusted values. …”

Image_1_Cannabinoid WIN55,212-2 reprograms monocytes and macrophages to inhibit LPS-induced inflammation.tif by Mario Pérez-Diego (14799367)

“…Mechanistically, WIN55,212-2 induced a metabolic and epigenetic shift in macrophages by decreasing LPS-induced mTORC1 signaling, commitment to glycolysis and active histone marks in pro-inflammatory cytokine promoters. …”

Image_7_Cannabinoid WIN55,212-2 reprograms monocytes and macrophages to inhibit LPS-induced inflammation.tif by Mario Pérez-Diego (14799367)

“…Mechanistically, WIN55,212-2 induced a metabolic and epigenetic shift in macrophages by decreasing LPS-induced mTORC1 signaling, commitment to glycolysis and active histone marks in pro-inflammatory cytokine promoters. …”

Table_1_Cannabinoid WIN55,212-2 reprograms monocytes and macrophages to inhibit LPS-induced inflammation.docx by Mario Pérez-Diego (14799367)

“…Mechanistically, WIN55,212-2 induced a metabolic and epigenetic shift in macrophages by decreasing LPS-induced mTORC1 signaling, commitment to glycolysis and active histone marks in pro-inflammatory cytokine promoters. …”

Image_6_Cannabinoid WIN55,212-2 reprograms monocytes and macrophages to inhibit LPS-induced inflammation.tif by Mario Pérez-Diego (14799367)

“…Mechanistically, WIN55,212-2 induced a metabolic and epigenetic shift in macrophages by decreasing LPS-induced mTORC1 signaling, commitment to glycolysis and active histone marks in pro-inflammatory cytokine promoters. …”

Combined B, T and NK Cell Deficiency Accelerates Atherosclerosis in BALB/c Mice by Fei Cheng (315348)

“…Increased atherosclerotic lesion development in C-<i>Ldlr</i>^<i>-/-</i> <i>Rag1</i>^<i>-/-</i> <i>Il2rg</i>^<i>-/-</i> triple mutants was associated with significantly increased numbers of macrophages and significantly decreased numbers of smooth muscle cells compared to both C-<i>Ldlr</i>^<i>-/-</i> wild type and C-<i>Ldlr</i>^<i>-/-</i> <i>Rag1</i>^<i>-/-</i> double mutants pointing to a plaque destabilizing effect of NK cell loss. …”

Image_3_Cannabinoid WIN55,212-2 reprograms monocytes and macrophages to inhibit LPS-induced inflammation.tif by Mario Pérez-Diego (14799367)

“…Mechanistically, WIN55,212-2 induced a metabolic and epigenetic shift in macrophages by decreasing LPS-induced mTORC1 signaling, commitment to glycolysis and active histone marks in pro-inflammatory cytokine promoters. …”

Hyperactivation of Wnt signaling and inactivation of Rb induced synergistic cell death effect in mammalian cells. by Tianyi Zhang (87298)

“…<p>In Rb mutant Du145 cells, knockdown of APC strongly reduced the level of APC protein detected by antibody staining (A) and increased the Wnt signaling reporter activity detected with the TOP-FOP luciferase assay (B). …”

AMD3100 prevented severe pulmonary arterial hypertension (PAH) in the SU5416/chronic hypoxia (SuHx) model. by Daniela Farkas (334698)

“…<p>(<b>A</b>) Representative von Willebrand Factor (vWF) immunohistochemistry indicates the occlusion of pulmonary arteries (arrows). …”

Monitoring changes in the ratio of the U1-70k isoforms in charge states of the intact spectrum and dissociation products. by Helena Hernández (259228)

First trimester maternal plasma proteomic changes predictive of spontaneous moderate/late preterm delivery by Danai Mavreli (16517629)

“…</p> <p>Isobaric tags for relative and absolute quantification (iTRAQ) coupled with LC–MS/MS was used for the analysis of five first trimester maternal plasma samples obtained from women with a subsequent moderate/late preterm sPTD and five women with term deliveries. …”