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step decrease » sizes decrease (Expand Search), teer decrease (Expand Search)
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a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
a step » _ step (Expand Search)
step decrease » sizes decrease (Expand Search), teer decrease (Expand Search)
we decrease » _ decrease (Expand Search), nn decrease (Expand Search), teer decrease (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide
Published 2016“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”
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12329
Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide
Published 2016“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”
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12330
Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide
Published 2016“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”
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12331
Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide
Published 2016“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”
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