Ba₂NaClP₂O₇: Unprecedented Phase Matchability Induced by Symmetry Breaking and Its Unique Fresnoite-Type Structure by Jie Chen (5892)

“…Herein, we report a unique Ba₂NaClP₂O₇ representing the first phase-matchable example of the fresnoite family, which exhibits excellent NLO properties, including short absorption edge (<176 nm, ca. 7.0 eV), strong second-harmonic generations at the incident lasers of 850–1110 nm (obv. 0.9–1.3 × KDP; cal. …”

Optical Phenomena and Antifrosting Property on Biomimetics Slippery Fluid-Infused Antireflective Films via Layer-by-Layer Comparison with Superhydrophobic and Antireflective Films by Kengo Manabe (1437730)

“…In particular, slippery liquid-infused surfaces are receiving remarkable interest as transparent, nonfouling, and antifrosting synthetic surfaces for solar cells and optical devices. Here we focus on the transparency of lubricant-infused texture on antireflective films fabricated by layer-by-layer self-assembly that decrease light scattering, which is important to maintain device properties. …”

Rab27A deficiency caused decreased amylase release from isolated acini. by Yanan Hou (736195)

SgG2 interacts with NGF. by Jorge Rubén Cabrera (685511)

Corticosterone treatment decreases MMP-2 promoter activity. by Eric A. Shikatani (131260)

“…<p>Endothelial cells (1.0×10⁶ cells) were plated in 35 mm² dishes coated with type I collagen and treated with 600 nM corticosterone for 48 hours. …”

TGFβ1 and COL1A1 expression was decreased and RASAL1 expression was increased by Western blotting in NTM cells under both normoxic and hypoxic conditions. by Fiona McDonnell (2636635)

“…NTM cells were treated with 0.3μM 5-aza for 24 hours and then subjected to 1%O₂ for the last 6 hours of treatment. We found that COL1A1 protein was decreased in the presence of 5-aza. …”

A Two-Year Randomized Trial of Interventions to Decrease Stress Hormone Vasopressin Production in Patients with Meniere’s Disease—A Pilot Study by Tadashi Kitahara (702040)

“…Because mental/physical stress and subsequent increase in the stress hormone vasopressin supposedly trigger Meniere's disease, we set a pilot study to seek new therapeutic interventions, namely management of vasopressin secretion, to treat this disease. …”

Substance P decreases PRL immunoreactivity in human epidermis. by Ewan A. Langan (405804)

Glycine is able to induce both a motility speed in- and decrease during zebrafish neuronal migration by Ulrike Theisen (4734555)

Choroidal Neovascularization Is Inhibited in Splenic-Denervated or Splenectomized Mice with a Concomitant Decrease in Intraocular Macrophage by Xue Tan (2994324)

5-FU induces a decrease in the total cell number in cultures of smooth muscle cells. by Marcelo de Carvalho Filgueiras (408321)

“…We found an initial reduction of 20% at 0.01 mM up to a reduction of 70% in 10 mM of 5-FU. …”

Image_3_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.TIF by Emilie Lesport (19214344)

“…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”

Table_1_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.DOCX by Emilie Lesport (19214344)

“…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”

Image_2_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.TIF by Emilie Lesport (19214344)

“…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”

Video_2_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.AVI by Emilie Lesport (19214344)

“…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”

Video_3_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.AVI by Emilie Lesport (19214344)

“…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”

Image_1_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.TIF by Emilie Lesport (19214344)

“…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”

Video_1_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.AVI by Emilie Lesport (19214344)

“…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”

Table_2_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.docx by Emilie Lesport (19214344)

“…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”

ACT-induced decrease of macrophage adhesion. by César Martín (229306)