Insulator-on-Conductor Fouling Amplifies Aqueous Electrolysis Rates by Harry Morris Rodriguez (18331235)

“…However, most organic molecules have low aqueous solubility, causing green and cheap electricity-driven reactions to suffer from intrinsically low reaction rates in industry’s solvent of choice: water. Here, we show that a strategic, partial electrode fouling with hydrophobic insulators (oils and plastics) offsets kinetic limitations caused by poor reactant solubility, opening a new path for the direct integration of renewable electricity into the production of commodity chemicals. …”

Insulator-on-Conductor Fouling Amplifies Aqueous Electrolysis Rates by Harry Morris Rodriguez (18331235)

“…However, most organic molecules have low aqueous solubility, causing green and cheap electricity-driven reactions to suffer from intrinsically low reaction rates in industry’s solvent of choice: water. Here, we show that a strategic, partial electrode fouling with hydrophobic insulators (oils and plastics) offsets kinetic limitations caused by poor reactant solubility, opening a new path for the direct integration of renewable electricity into the production of commodity chemicals. …”

Breathable, Wireless, Thin-Film Wearable Biopatch Using Noise-Reduction Mechanisms by Nathan Rodeheaver (11939515)

“…Motion artifacts are also intensified during prolonged and repeated use from factors like decreased adhesion by sweating or dust. Here, we introduce a breathable, wireless wearable biopatch using an enhanced noise-reduction mechanism. …”

Breathable, Wireless, Thin-Film Wearable Biopatch Using Noise-Reduction Mechanisms by Nathan Rodeheaver (11939515)

“…Motion artifacts are also intensified during prolonged and repeated use from factors like decreased adhesion by sweating or dust. Here, we introduce a breathable, wireless wearable biopatch using an enhanced noise-reduction mechanism. …”

Breathable, Wireless, Thin-Film Wearable Biopatch Using Noise-Reduction Mechanisms by Nathan Rodeheaver (11939515)

“…Motion artifacts are also intensified during prolonged and repeated use from factors like decreased adhesion by sweating or dust. Here, we introduce a breathable, wireless wearable biopatch using an enhanced noise-reduction mechanism. …”

Data_Sheet_1_Effect of cAMP Receptor Protein Gene on Growth Characteristics and Stress Resistance of Haemophilus parasuis Serovar 5.docx by Changsheng Jiang (2277841)

“…In order to investigate the function of the crp gene in the growth characteristics of H. parasuis serovar 5 (HPS5) and its ability to overcome adverse environmental stresses, a crp mutant strain (Δcrp) was constructed and verified. …”

Table_1_Ideal Cardiovascular Health in Young Adults With Established Cardiovascular Diseases.DOCX by Jane A. Leopold (12098417)

“…Young adults with cardiovascular disease (n = 349) were older, had a lower socioeconomic status, a higher prevalence of risk factors, and lower Life's Simple 7 Health Scores (6.4 ± 1.5 vs. 7.1 ± 1.5, p < 0.01) compared to young adults without cardiovascular disease (n = 696). …”

Image_1_CircKIF5B Promotes Hepatocellular Carcinoma Progression by Regulating the miR-192 Family/XIAP Axis.tif by Zhenghua Fei (3514445)

“…Moreover, we found that silencing circKIF5B markedly reduced the proliferation, invasion, and metastasis of liver cancer cells by sponging the miR-192 family, thus decreasing the expression of X-linked inhibitor of apoptosis (XIAP).…”

Image_2_CircKIF5B Promotes Hepatocellular Carcinoma Progression by Regulating the miR-192 Family/XIAP Axis.tif by Zhenghua Fei (3514445)

“…Moreover, we found that silencing circKIF5B markedly reduced the proliferation, invasion, and metastasis of liver cancer cells by sponging the miR-192 family, thus decreasing the expression of X-linked inhibitor of apoptosis (XIAP).…”

Image_3_CircKIF5B Promotes Hepatocellular Carcinoma Progression by Regulating the miR-192 Family/XIAP Axis.tif by Zhenghua Fei (3514445)

“…Moreover, we found that silencing circKIF5B markedly reduced the proliferation, invasion, and metastasis of liver cancer cells by sponging the miR-192 family, thus decreasing the expression of X-linked inhibitor of apoptosis (XIAP).…”

Table_1_CircKIF5B Promotes Hepatocellular Carcinoma Progression by Regulating the miR-192 Family/XIAP Axis.docx by Zhenghua Fei (3514445)

“…Moreover, we found that silencing circKIF5B markedly reduced the proliferation, invasion, and metastasis of liver cancer cells by sponging the miR-192 family, thus decreasing the expression of X-linked inhibitor of apoptosis (XIAP).…”

Image_4_CircKIF5B Promotes Hepatocellular Carcinoma Progression by Regulating the miR-192 Family/XIAP Axis.tif by Zhenghua Fei (3514445)

“…Moreover, we found that silencing circKIF5B markedly reduced the proliferation, invasion, and metastasis of liver cancer cells by sponging the miR-192 family, thus decreasing the expression of X-linked inhibitor of apoptosis (XIAP).…”

Table_5_IRX3 plays an important role in the pathogenesis of metabolic-associated fatty liver disease by regulating hepatic lipid metabolism.xlsx by Yongqiang Ma (484555)

“…<p>Metabolic-associated fatty liver disease (MAFLD) affects approximately a quarter of the global population. Identification of the key genes and pathways involved in hepatic lipid metabolism is of the utmost importance for the diagnosis, treatment, and prevention of MAFLD. …”

Image 5_Isolation and identification of Alternaria alstroemeriae causing postharvest black rot in citrus and its control using curcumin-loaded nanoliposomes.jpeg by Jie Li (15030)

“…In order to further elucidate the pathogen of citrus fruit rot in Yunnan Province, the pathogenic fungi causing citrus fruit rot were identified through isolation and purification, pathogenicity testing, morphological characteristics, and rDNA ITS sequence analysis. Meanwhile, we synthesized curcumin-loaded nanoliposomes, a potential management approach to control citrus postharvest pathogen, and conducted vitro and vivo experiment to investigate the effects of different curcumin-loaded nanoliposomes treatments inhibitory effect to pathogen A. alstroemeriae. …”

Video_2_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Single molecule analysis of TRPV4 channels showed that the density of TRPV4 at the plasma membrane is controlled through two modes of membrane trafficking, complete, and partial vesicular fusion. Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_4_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Single molecule analysis of TRPV4 channels showed that the density of TRPV4 at the plasma membrane is controlled through two modes of membrane trafficking, complete, and partial vesicular fusion. Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_3_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Single molecule analysis of TRPV4 channels showed that the density of TRPV4 at the plasma membrane is controlled through two modes of membrane trafficking, complete, and partial vesicular fusion. Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_6_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Single molecule analysis of TRPV4 channels showed that the density of TRPV4 at the plasma membrane is controlled through two modes of membrane trafficking, complete, and partial vesicular fusion. Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_1_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Single molecule analysis of TRPV4 channels showed that the density of TRPV4 at the plasma membrane is controlled through two modes of membrane trafficking, complete, and partial vesicular fusion. Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Image_1_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.pdf by Sara Baratchi (357850)

“…Single molecule analysis of TRPV4 channels showed that the density of TRPV4 at the plasma membrane is controlled through two modes of membrane trafficking, complete, and partial vesicular fusion. Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”