Table 2_Integrated machine learning analysis of 30 cell death patterns identifies a novel prognostic signature in glioma.xlsx حسب Minhao Huang (4952764)

"…A pan-death prognostic signature (Cell-Death Score, CDS) was constructed using 114 machine learning algorithm combinations, refined via CoxBoost to select 25 key genes. …"

Table 1_Integrated machine learning analysis of 30 cell death patterns identifies a novel prognostic signature in glioma.xlsx حسب Minhao Huang (4952764)

"…A pan-death prognostic signature (Cell-Death Score, CDS) was constructed using 114 machine learning algorithm combinations, refined via CoxBoost to select 25 key genes. …"

Image 3_Integrated machine learning analysis of 30 cell death patterns identifies a novel prognostic signature in glioma.jpeg حسب Minhao Huang (4952764)

"…A pan-death prognostic signature (Cell-Death Score, CDS) was constructed using 114 machine learning algorithm combinations, refined via CoxBoost to select 25 key genes. …"

Image 2_Integrated machine learning analysis of 30 cell death patterns identifies a novel prognostic signature in glioma.jpeg حسب Minhao Huang (4952764)

"…A pan-death prognostic signature (Cell-Death Score, CDS) was constructed using 114 machine learning algorithm combinations, refined via CoxBoost to select 25 key genes. …"

Table 3_Integrated machine learning analysis of 30 cell death patterns identifies a novel prognostic signature in glioma.xlsx حسب Minhao Huang (4952764)

"…A pan-death prognostic signature (Cell-Death Score, CDS) was constructed using 114 machine learning algorithm combinations, refined via CoxBoost to select 25 key genes. …"

Image 1_Integrated machine learning analysis of 30 cell death patterns identifies a novel prognostic signature in glioma.jpeg حسب Minhao Huang (4952764)

"…A pan-death prognostic signature (Cell-Death Score, CDS) was constructed using 114 machine learning algorithm combinations, refined via CoxBoost to select 25 key genes. …"

Data Sheet 1_Impaired and restored CD8+ T cell responses to a sequentially mutated spike epitope in Omicron variants.docx حسب Eun Joo Chung (9678137)

"…Our findings suggest that LYN may function both as an immune-evasive hotspot and as a cross-reactive epitope, underscoring the importance of continued epitope-level surveillance as future variants emerge.…"

DataSheet1_Identification of novel markers for neuroblastoma immunoclustering using machine learning.zip حسب Longguo Zhang (20105238)

"…Three machine learning algorithms (LASSO, SVM-RFE, and Random Forest) were used to screen biomarkers and synthesize their function in neuroblastoma.…"

Data Sheet 1_Resveratrol contributes to NK cell-mediated breast cancer cytotoxicity by upregulating ULBP2 through miR-17-5p downmodulation and activation of MINK1/JNK/c-Jun signali... حسب Bisha Ding (5803799)

"…UL16-binding protein 2 (ULBP2), always expressed or elevated on cancer cells, functions as a key NKG2D ligand. ULBP2-NKG2D ligation initiates NK cell activation and subsequent targeted elimination of cancer cells. …"

Table 12_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.xlsx حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 9_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.xlsx حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 8_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.xlsx حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 1_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.xlsx حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 2_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.xlsx حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 3_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.xlsx حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 5_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.doc حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 13_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.doc حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 6_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.xlsx حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 11_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.xlsx حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"

Table 7_Decoding immune-metabolic crosstalk in ARDS: a transcriptomic exploration of biomarkers, cellular dynamics, and therapeutic pathways.xlsx حسب Ting Wu (106368)

"…RT-qPCR confirmed a significant upregulation of SMARCD3 and TCN1 in ARDS samples, aligning with dataset expression analysis results. Both in vitro and in vivo experiments demonstrated that modulation of SMARCD3 and TCN1 (but not RPL14) significantly affected mitochondrial function, oxidative stress, apoptosis, glucose metabolism and inflammatory cytokine expression.…"