Showing 81 - 100 results of 31,445 for search '(( brain ((((a decrease) OR (we decrease))) OR (greater increase)) ) OR ( a greater increase ))', query time: 0.44s Refine Results
  1. 81

    Data_Sheet_1_Age-Related Decrease in Default-Mode Network Functional Connectivity Is Accelerated in Patients With Major Depressive Disorder.docx by Shixiong Tang (6014612)

    Published 2022
    “…However, little is known whether such process would be reflected by a more rapid aging of the brain function. In this study, we tested the hypothesis that MDD would be characterized by accelerated aging of the brain’s default-mode network (DMN) functions. …”
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    Decreased brain-derived neurotrophic factor plasma levels in psoriasis patients by A.R. Brunoni (5694239)

    Published 2019
    “…Our finding, that BDNF is decreased in psoriasis, supports the concept of a brain-skin connection in psoriasis. …”
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    A Platinum(IV) ProdrugPerfluoroaryl Macrocyclic Peptide Conjugate Enhances Platinum Uptake in the Brain by Colin M. Fadzen (4543159)

    Published 2020
    “…We investigate the pharmacokinetics of this conjugate in mice and show that the amount of platinum in the brain after treatment with the conjugate is 15-fold greater than with cisplatin after 5 h.…”
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    DataSheet_1_Nocturnal melatonin increases glucose uptake via insulin-independent action in the goldfish brain.pdf by Kazuki Watanabe (519518)

    Published 2023
    “…Using an insulin-free medium, we demonstrated that melatonin treatment increased glucose uptake in a dose-dependent manner in primary cell cultures of goldfish brain and liver cells. …”
  14. 94

    VPC and stare controls are readily decodable on a similar time-frame as changes in consciousness, and represent a clear shift in the relative importance of different frequencies. by Michelle J. Redinbaugh (13029044)

    Published 2022
    “…<p><b>A</b>, Decoding accuracy (±SE) across time relative to event onset of VPC and stare controls using 25 frequency (δ, θ, α, β, γ) by brain area (CN, Ld, Ls, Fd, Fs) features. …”
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    Custom MATLAB code. by Samuel A. Jones (17912769)

    Published 2024
    “…This dissociation—between comparable information encoded in brain activation patterns across the 2 age groups, but age-related increases in regional blood-oxygen-level-dependent responses—contradicts the widespread notion that older adults recruit new regions as a compensatory mechanism to encode task-relevant information. …”
  17. 97

    Mass univariate fMRI analysis—Results. by Samuel A. Jones (17912769)

    Published 2024
    “…This dissociation—between comparable information encoded in brain activation patterns across the 2 age groups, but age-related increases in regional blood-oxygen-level-dependent responses—contradicts the widespread notion that older adults recruit new regions as a compensatory mechanism to encode task-relevant information. …”
  18. 98

    Image_3_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.TIF by Emilie Lesport (19214344)

    Published 2024
    “…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”
  19. 99

    Table_1_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.DOCX by Emilie Lesport (19214344)

    Published 2024
    “…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”
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    Image_2_A decrease in Fkbp52 alters autophagosome maturation and A152T-tau clearance in vivo.TIF by Emilie Lesport (19214344)

    Published 2024
    “…<p>The failure of the autophagy-lysosomal pathway to clear the pathogenic forms of Tau exacerbates the pathogenesis of tauopathies. We have previously shown that the immunophilin FKBP52 interacts both physically and functionally with Tau, and that a decrease in FKBP52 protein levels is associated with Tau deposition in affected human brains. …”