Search alternatives:
marked decrease » marked increase (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
26 a » 26 0 (Expand Search), 2 a (Expand Search), 26 c (Expand Search)
marked decrease » marked increase (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
26 a » 26 0 (Expand Search), 2 a (Expand Search), 26 c (Expand Search)
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Loss of MALS-1 function suppresses the mitochondrial and axon degeneration phenotypes that are caused by loss of RBM-26 function.
Published 2024“…(D) Loss of RBM-26 protein expression causes a decrease in mitoribosome expression, which was assayed with the MitoRibo-Tag system consisting of the MRPL-58 mitoribosomal protein tagged at its C-terminus with 3XFLAG (MRPL-58::3XFLAG). …”
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Underlying data for the main figures.
Published 2024“…Autism-associated missense variants in RBM-26 cause a sharp decrease in RBM-26 protein expression along with defects in axon overlap and axon degeneration that occurs during larval development. …”
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Underlying data for the supplemental figures.
Published 2024“…Autism-associated missense variants in RBM-26 cause a sharp decrease in RBM-26 protein expression along with defects in axon overlap and axon degeneration that occurs during larval development. …”
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Overexpression of MALS-1 reduces mitochondria in the PLM axon and causes axon degeneration and axon overlap defects.
Published 2024“…<p>(A–C) Overexpressed MALS-1::Scarlet protein colocalizes with mitochondria. …”
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