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therapeutic resistance » therapeutic research (Expand Search), therapeutic response (Expand Search)
decrease therapeutic » increased therapeutic (Expand Search), degrader therapeutics (Expand Search), great therapeutic (Expand Search)
therapeutic resistance » therapeutic research (Expand Search), therapeutic response (Expand Search)
decrease therapeutic » increased therapeutic (Expand Search), degrader therapeutics (Expand Search), great therapeutic (Expand Search)
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Genomic driver underlies clonal shift of methicillin-resistant <i>Staphylococcus aureus</i> in the healthcare setting
Published 2025“…<p>The epidemiology of healthcare-associated methicillin-resistant <i>Staphylococcus aureus</i> (HA-MRSA) has shifted in recent decades, with a notable decline in the lineage ST239 and continued dominance of ST5. …”
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Metformin and ZLN005 prevent NTM-mediated macrophage dysfunction through PGC-1α.
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Metformin and ZLN005 attenuate MAC-mediated mitochondrial membrane potential damage.
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Metformin and ZLN005 facilitate increased phagocytic uptake and intracellular killing of MAB.
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NTM infection induces mitochondrial damage and reduces mitochondria number.
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Excel raw data.
Published 2025“…The effectiveness of currently available antimicrobial is decreasing due to the increasing prevalence of resistant strains among bacterial isolates. …”
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The raw data of the paper with the title of " The Protective Effect of Schisandrin C against Methicillin-Resistant Staphylococcus aureus - Induced Otitis Media "
Published 2025“…In rat models of MRSA-induced AOM and CSOM, Sch C significantly reduced bacterial load and inflammatory responses by decreasing proinflammatory cytokine levels. …”
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Table 1_IGFBP5 mediates the therapeutic effect of isoliquiritigenin in myocardial ischemia-reperfusion injury via AKT/GLUT4 regulated insulin resistance.xlsx
Published 2025“…</p>Propose<p>In this study, we aimed to investigate ISL’s therapeutic effects on MIRI. Moreover, we elucidate the underlying mechanisms of ISL regulated myocardium insulin resistance in vivo and in vitro.…”
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Combination therapy exhibited apoptosis in Ba/F3 EML4-ALKmutant cell models.
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Ba/F3 EML4-ALK<sup>mutants</sup> exhibit variable dose-response to ABT-199 treatment.
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Molecular docking showed that ABT-199 can fit into active site of ALK<sup>Mutant</sup>.
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