Impacts of conventional and conservation crop rotations (2017–2019 and 2019–2020) for the land management (ha.year) functional unit. by Maria Vittoria Guidoboni (15852669)

V5-SgG2 transfection in the epidermis modifies the termination site of peptidergic FNE. by Jorge Rubén Cabrera (685511)

Concentration-dependent effect of pre-exposure with rotenone (method No. 2). by Joseph A. Covi (3131166)

Carbon (C) balance per crop and its components for wheat and popcorn estimated by the AMG model: Organic C input from cash-crop residues (Ccrop), mineralisation of soil C (Cmin), o... by Maria Vittoria Guidoboni (15852669)

Decrease or lack of decoration of <i>L</i>. <i>infantum</i> LEM 5700 parasites with GTPase Rab7, Lamp-2, and Qb-SNAREs Vti1a and Vti1b in macrophage-like RAW 264.7 cells infected i... by Sandrine Cojean (274005)

“…<p><b>(A)</b><i>z</i>-stack and 3D-reconstruction surface rendering confocal micrographs showing decreased decoration of fusiform-like and ellipsoidal parasites hosted in Retro-2 (1 μM)-treated cells with Rab7-positive patches. …”

Cullin-1/5 are required for NF-κB signaling during <i>Eh</i>-macrophage contact. by Attinder Chadha (11414273)

“…<p><b>(A)</b> THP-1 cells were transfected with 100nM of cullin-1 siRNA and scrambled control (SC siRNA) and in <b>(B)</b> 100nM of cullin-5 siRNA and scrambled control (SC siRNA). …”

Associations between Baseline Sleep Quality and Average Screen-Time. by Matthew A. Christensen (3339732)

CTV p20 interacts with autophagy-related proteins NbATG8a and NbATG8f and induces autophagy. by Yongle Zhang (11355546)

Mice infected with cMRSA post H1N1 have higher bacterial burden as compared to virus infected animals alone and treatment with antibiotics post bacterial challenge decreases bacter... by Urvashi Bhan (247436)

Figure 1 from Gut Microbiome Profiling in Eμ-TCL1 Mice Reveals Intestinal Changes and a Dysbiotic Signature Specific to Chronic Lymphocytic Leukemia by Sydney A. Skupa (18597728)

“…Hashtags denote the significance between WT B6 and Eμ-TCL1 mice at each time point (^##, <i>P</i> < 0.01; ^###, <i>P</i> < 0.001). …”

Image_6_Resting Energy Expenditure and Body Composition in Children and Adolescents With Genetic, Hypothalamic, Medication-Induced or Multifactorial Severe Obesity.tif by Ozair Abawi (8816267)

“…Mean REE% was higher in children with non-syndromic genetic obesity (107.4% ± 12.7) and lower in children with hypothalamic obesity (87.6% ± 14.2) compared to multifactorial obesity (100.5% ± 12.6, both p<0.01). In 9 children with pseudohypoparathyroidism type 1a, mean REE% was similar (100.4 ± 5.1). …”

Image_3_Resting Energy Expenditure and Body Composition in Children and Adolescents With Genetic, Hypothalamic, Medication-Induced or Multifactorial Severe Obesity.tif by Ozair Abawi (8816267)

“…Mean REE% was higher in children with non-syndromic genetic obesity (107.4% ± 12.7) and lower in children with hypothalamic obesity (87.6% ± 14.2) compared to multifactorial obesity (100.5% ± 12.6, both p<0.01). In 9 children with pseudohypoparathyroidism type 1a, mean REE% was similar (100.4 ± 5.1). …”

Image_8_Resting Energy Expenditure and Body Composition in Children and Adolescents With Genetic, Hypothalamic, Medication-Induced or Multifactorial Severe Obesity.tif by Ozair Abawi (8816267)

“…Mean REE% was higher in children with non-syndromic genetic obesity (107.4% ± 12.7) and lower in children with hypothalamic obesity (87.6% ± 14.2) compared to multifactorial obesity (100.5% ± 12.6, both p<0.01). In 9 children with pseudohypoparathyroidism type 1a, mean REE% was similar (100.4 ± 5.1). …”

Image_7_Resting Energy Expenditure and Body Composition in Children and Adolescents With Genetic, Hypothalamic, Medication-Induced or Multifactorial Severe Obesity.tif by Ozair Abawi (8816267)

“…Mean REE% was higher in children with non-syndromic genetic obesity (107.4% ± 12.7) and lower in children with hypothalamic obesity (87.6% ± 14.2) compared to multifactorial obesity (100.5% ± 12.6, both p<0.01). In 9 children with pseudohypoparathyroidism type 1a, mean REE% was similar (100.4 ± 5.1). …”

Image_4_Resting Energy Expenditure and Body Composition in Children and Adolescents With Genetic, Hypothalamic, Medication-Induced or Multifactorial Severe Obesity.tif by Ozair Abawi (8816267)

“…Mean REE% was higher in children with non-syndromic genetic obesity (107.4% ± 12.7) and lower in children with hypothalamic obesity (87.6% ± 14.2) compared to multifactorial obesity (100.5% ± 12.6, both p<0.01). In 9 children with pseudohypoparathyroidism type 1a, mean REE% was similar (100.4 ± 5.1). …”

Image_5_Resting Energy Expenditure and Body Composition in Children and Adolescents With Genetic, Hypothalamic, Medication-Induced or Multifactorial Severe Obesity.tif by Ozair Abawi (8816267)

“…Mean REE% was higher in children with non-syndromic genetic obesity (107.4% ± 12.7) and lower in children with hypothalamic obesity (87.6% ± 14.2) compared to multifactorial obesity (100.5% ± 12.6, both p<0.01). In 9 children with pseudohypoparathyroidism type 1a, mean REE% was similar (100.4 ± 5.1). …”

Image_1_Resting Energy Expenditure and Body Composition in Children and Adolescents With Genetic, Hypothalamic, Medication-Induced or Multifactorial Severe Obesity.tif by Ozair Abawi (8816267)

“…Mean REE% was higher in children with non-syndromic genetic obesity (107.4% ± 12.7) and lower in children with hypothalamic obesity (87.6% ± 14.2) compared to multifactorial obesity (100.5% ± 12.6, both p<0.01). In 9 children with pseudohypoparathyroidism type 1a, mean REE% was similar (100.4 ± 5.1). …”

DataSheet_1_Resting Energy Expenditure and Body Composition in Children and Adolescents With Genetic, Hypothalamic, Medication-Induced or Multifactorial Severe Obesity.docx by Ozair Abawi (8816267)

“…Mean REE% was higher in children with non-syndromic genetic obesity (107.4% ± 12.7) and lower in children with hypothalamic obesity (87.6% ± 14.2) compared to multifactorial obesity (100.5% ± 12.6, both p<0.01). In 9 children with pseudohypoparathyroidism type 1a, mean REE% was similar (100.4 ± 5.1). …”

Image_2_Resting Energy Expenditure and Body Composition in Children and Adolescents With Genetic, Hypothalamic, Medication-Induced or Multifactorial Severe Obesity.tif by Ozair Abawi (8816267)

“…Mean REE% was higher in children with non-syndromic genetic obesity (107.4% ± 12.7) and lower in children with hypothalamic obesity (87.6% ± 14.2) compared to multifactorial obesity (100.5% ± 12.6, both p<0.01). In 9 children with pseudohypoparathyroidism type 1a, mean REE% was similar (100.4 ± 5.1). …”

Integration of Metabolomics, Transcriptomics, and 16s rRNA Sequencing Reveals the Mechanism of Morus alba <i>L.</i> (Sangzhi) Alkaloids (SZ-A) in Improving Cholesterol Metabolism i... by Caina Li (4126462)

“…In this study, SZ-A was orally administered to Zucker diabetic fatty rats at doses of 100 and 200 mg/kg once daily for 9 weeks. …”