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1541
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1542
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1543
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1544
All data points from Fig 2.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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1545
All data points from Fig 8.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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1546
All data points from Fig 3.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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1547
All data points from Fig 1.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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1548
All data points from Fig 4.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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1549
All data points from Fig 9.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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1550
All data points from Fig 7.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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1551
All data points from Fig 6.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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1552
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1553
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1554
Simultaneously inhibition <i>Nlu-miR-34</i> and <i>NlInR2</i> in SW strain induced the decrease of SW BPHs.
Published 2019“…<p>(A) Antagomir-34 (with two different quantities) and ds<i>NlInR2</i> (0.84 ng) were injected into the third instar SW strain nymphs, showed that injection of antagomir-34 decreased the ratio of SW type BPH (Chi-square test, 40ng: χ<sup>2</sup> = 5.8, df = 1, <i>p</i> = 0.003; 60ng: χ<sup>2</sup> = 7.9, df = 1, <i>p</i> = 0.042). …”
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1555
Severe decreases of TECs and impaired TEC expansion in the absence of mTORC1/Raptor.
Published 2016“…Each circle or square represents one WT or <i>Rpt</i><sup><i>f/f</i></sup><i>-Foxn1Cre</i> mouse, respectively. *, <i>p</i> < 0.05; **, <i>p</i> < 0.01; ***, <i>p</i> < 0.001 determined by two-tail Student’s <i>t</i> test.…”
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1556
Decreased self-renewal capacity associated with senescence was prevented in human MSCs following treatment with Ki16425, an LPA<sub>1</sub>/LPA<sub>3</sub> antagonist.
Published 2012“…Human MSCs at passage 2 were cultured in the presence or absence of Ki16425 for the indicated days prior to cell lysis. Fold-change represents decrease in band intensity of Ki16425 treatment for 18 days compared with a control treatment for the same period of time. …”
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1557
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1558
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1559
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1560