Search alternatives:
point decrease » point increase (Expand Search)
nn decrease » _ decrease (Expand Search), mean decrease (Expand Search), gy decreased (Expand Search)
e decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
point decrease » point increase (Expand Search)
nn decrease » _ decrease (Expand Search), mean decrease (Expand Search), gy decreased (Expand Search)
e decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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781
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782
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783
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784
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785
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786
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788
eNOS phosphorylation increased intracellular ROS generation and decreased cell viability in BH4-depleted cells.
Published 2015“…(B) VEGF-mediated eNOS phosphorylation resulted a significant ROS increase in eNOS overexpressing HEK 293 cells (n = 5 fields, *, <i>p</i> < 0.05). …”
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789
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790
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791
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792
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793
Interactions between E1A and PKA are required for full HAdV-5 progeny production.
Published 2016“…Growth of WT virus was decreased by knockdown of each PKA subunit. Growth of HAdV E1A Δ4–25 was not affected by knockdown of PKA regulatory subunits, but was affected by knockdown of PKA Cα (though to a lesser extent than WT HAdV-5). …”
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794
Supplementary Material for: Blood Outgrowth Endothelial Cells Reduce Hypoxia-Mediated Fibroblast to Myofibroblast Conversion by Decreasing Proangiogenic Cytokines
Published 2017“…<p><b><i>Purpose:</i></b> Hypoxic conditions cause fibroblasts to differentiate into alpha smooth-muscle cell actin (α -SMA)-positive cells, i.e. myofibroblasts. …”
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795
Decreasing prevalence of contamination with extended-spectrum beta-lactamase-producing <i>Enterobacteriaceae</i> (ESBL-E) in retail chicken meat in the Netherlands
Published 2019“…<div><p>Retail chicken meat is a potential source of extended-spectrum beta-lactamase-producing <i>Enterobacteriaceae</i> (ESBL-E). …”
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796
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797
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798
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799
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800