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fold decrease » fold increase (Expand Search), fold increased (Expand Search)
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841
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842
Significant decrease of SMAD4 protein in DBA iPSCs.
Published 2015“…A) Slight decrease of mRNA level of <i>SMAD4</i> in the DBA iPSCs with <i>RPS19</i> or <i>RPL5</i> mutations. …”
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843
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844
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845
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846
Change of <i>SPDEF</i>, <i>MUC5AC</i>, and <i>MUC16</i> mRNA expression levels at follow-up points after rebamipide instillation treatment in SS-DE patients.
Published 2020“…<p>Least-square means (LS means ± 95% confidence interval) ware plotted for relative expression levels of <i>SPDEF</i>, <i>MUC5AC</i>, and <i>MUC16</i> mRNA at each observation point. …”
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847
Decreased biomechanical stability of bones from <i>Col1a1-Krm2</i> transgenic mice.
Published 2013“…(D) Cortical thickness (C.Th.) and bone mineral density (vBMD) are decreased in <i>Col1a1-Krm2</i> transgenic mice compared to wildtype littermates. …”
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848
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849
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850
A R13A point mutation in C4 attenuates CLCuMuV infection.
Published 2018“…<b>(C)</b> The mutant CLCuMuV carrying C4<sup>R13A</sup> (CLCuMuV-C4<sup>R13A</sup>), which contains a R13A mutation in <i>C4</i>, showed the decreased viral symptom compared to wild type CLCuMuV. …”
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851
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852
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853
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854
Tun increases mitochondrial fission, inflammatory cell infiltration, and decreased ER-mitochondria interaction in neonatal rat lungs.
Published 2022“…The decreased expressions of GRP75 (0.5±0.2-fold, p = 0.006767, n = 5) and acyl-CoA synthetase long-chain family member 4 (ASCL4; 0.3±0.1-fold, p<0.001, n = 5) indicate a decreased interaction between ER and mitochondria by Tun. …”
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855
Nitrile in the Hole: Discovery of a Small Auxiliary Pocket in Neuronal Nitric Oxide Synthase Leading to the Development of Potent and Selective 2‑Aminoquinoline Inhibitors
Published 2017“…We previously developed a class of membrane-permeable 2-aminoquinoline inhibitors and later rearranged the scaffold to decrease off-target binding. …”
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856
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857
Effect of DFATs implantation on the inhibition of atrophy in the atrophic vocal fold muscle.
Published 2025Subjects: -
858
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859
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860