S1 Raw images - حسب Dequan Wang (6007007)

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shCCND1 plasmid. حسب Dequan Wang (6007007)

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CCND1 promoted chemoresistance in CRC. حسب Dequan Wang (6007007)

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RT-qPCR Primer. حسب Dequan Wang (6007007)

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CCND1 is a target gene of miR-1247-3p. حسب Dequan Wang (6007007)

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miR-1247-3p was downregulated in CRC resistant cells. حسب Dequan Wang (6007007)

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miR-1247-3p plasmid. حسب Dequan Wang (6007007)

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RNA-sequencing data screening for miR-1247-3p target genes. حسب Dequan Wang (6007007)

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Dual luciferase primer. حسب Dequan Wang (6007007)

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Overexpression of miR-1247-3p promoted apoptosis and inhibited resistance in drug-resistant cells. حسب Dequan Wang (6007007)

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Effect of miR-1247-3p on survival in colorectal cancer patients. حسب Dequan Wang (6007007)

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Introduction to the dataset. حسب Dequan Wang (6007007)

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Downregulation of miR-1247-3p inhibited apoptosis and reduced chemosensitivity in cells. حسب Dequan Wang (6007007)

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miR-1247-3p was downregulated in tissues of patients with CRC and was associated with prognosis and drug resistance. حسب Dequan Wang (6007007)

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Cinacalcet administered early in the inactive phase markedly decrease parathyroid Ki-67 index. حسب Søren Egstrand (10906087)

The parameters of gas exchange markedly decreased in maize <i>zmstomagen1/2</i> mutant compared with in wild type (WT). حسب Juan-Juan Xu (18590868)

Table 1_Previous treatment decreases efficacy of pralsetinib in RET fusion-positive non-small-cell lung cancer.doc حسب Lei Wang (6656)

"…The ARROW trial revealed that RET fusion-positive non-small-cell lung cancer (NSCLC) can benefit from pralsetinib with tolerable adverse events (AEs). …"

Madrasin decreases transcription of protein-coding genes. حسب Michael Tellier (8284752)

Madrasin decreases transcription of protein-coding genes. حسب Michael Tellier (8284752)

<b>The loss of insulin-positive cell clusters precedes the decrease of islet frequency and beta cell area in type 1 diabetes</b> حسب Denise M. Drotar (21679539)

"…Moreover, changes in endocrine composition also occurred in mAAb+ donors, including a significant decrease in the INS+ islet fraction. These data suggest preferential loss of INS+ small endocrine objects early in type 1 diabetes development.…"