Loss of MALS-1 function suppresses the mitochondrial and axon degeneration phenotypes that are caused by loss of RBM-26 function. by Tamjid A. Chowdhury (14149846)

“…For panel B, error bars represent the standard error of the mean (<i>n</i> = 25), and statistical significance was analyzed by one-way ANOVA with a Tukey post hoc test (*<i>p</i> < 0.05 and **<i>p</i> < 0.01). …”

Overexpression of MALS-1 reduces mitochondria in the PLM axon and causes axon degeneration and axon overlap defects. by Tamjid A. Chowdhury (14149846)

“…<p>(A–C) Overexpressed MALS-1::Scarlet protein colocalizes with mitochondria. …”

Loss of RBM-26 causes mitochondria dysfunction in PLM. by Tamjid A. Chowdhury (14149846)

RBM-26 is expressed in the PLM and functions within neurons to protect against axon degeneration and prevent axon overlap defects. by Tamjid A. Chowdhury (14149846)

Mutations in <i>rbm-26</i> cause axon degeneration and axon overlap defects. by Tamjid A. Chowdhury (14149846)

E (soil aggregate stability energy) is the amount of energy required when 99% of the soil is disrupted, Ecrit (soil critical stabilization energy) is the energy required when the i... by Xiaoxu Zhu (11764430)

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Baseline and Post-Treatment Myocardial Perfusion Defects Expressed as a Percentage of the Total Left Ventricular Surface Area in Each Experimental Group. by Káryta Suely Macêdo Martins (22404155)

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Synthesis and Biological Evaluation of MEK/mTOR Multifunctional Inhibitors as Novel Anticancer Agents by Marcian E. Van Dort (21374093)

“…Additionally, compound LP-65 demonstrated significant modulation of MEK and mTOR signaling activity in human glioma cells (D54) and human melanoma cells (A375), with a corresponding decrease in cellular proliferation and migration. …”

Decomposition analysis of the changes in prevalence (A), DALYs (B), and mortality (C) rates of IBD among women across global, 5 SDI and 21 GBD regions. by Jiefeng Zhao (9090305)

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<i>Kif15</i> deficiency affects the expression of excitatory postsynaptic receptors in PFC and hippocampus. by Xiaomei He (795333)

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Characteristics of study subjects. by Jung Eun Choi (563339)

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<i>Kif15</i> deficient mice exhibit both depressive and manic behaviors in adulthood. by Xiaomei He (795333)

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The regulation of LGG supplementation of the adaptive immune triggered by <i>M</i>. <i>pneumoniae</i>. by Huanbing Long (19949784)

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Intrauterine supplementation of <i>Kif15</i> in E15 <i>Kif15</i>^<i>-/-</i> mice rescued the distribution of PSD95 in <i>Kif15</i>^<i>-/-</i> PFC neurons. by Xiaomei He (795333)

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KIF15 interacts with PSD95 and affect its localization in neuronal cytoplasm neurons of the PFC. by Xiaomei He (795333)

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<i>Kif15</i> deficiency affects the expression of Gephyrin and GABRB1 in PFC and hippocampus. by Xiaomei He (795333)

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The knockdown efficiency of KIF15 in 293T cells was assessed. by Xiaomei He (795333)

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Supplementation of five Lactobacillus strains affected the colonization of <i>M</i>. <i>pneumoniae</i> and lung injuries. by Huanbing Long (19949784)

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Gene knockout strategy and genotype identification in KO mice. by Xiaomei He (795333)

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Both live and heat-inactivated LGG supplementation attenuated lung tissue injury caused by <i>M</i>. <i>pneumoniae</i> infection. by Huanbing Long (19949784)

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