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Showing 181 - 200 results of 21,342 for search '(( significant ((small decrease) OR (fold decrease)) ) OR ( significant decrease decrease ))', query time: 0.61s Refine Results
  1. 181
    The growth of co-transformed cells in the peritoneal cavity alters their cellular gene expression to mirror that of PEL biopsies.

    The growth of co-transformed cells in the peritoneal cavity alters their cellular gene expression to mirror that of PEL biopsies. by Mitchell Hayes (5334887)

    Published 2025
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  2. 182
    Co-infected cells express both KSHV and EBV genes <i>in vitro</i> necessary for their survival and growth.

    Co-infected cells express both KSHV and EBV genes <i>in vitro</i> necessary for their survival and growth. by Mitchell Hayes (5334887)

    Published 2025
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  3. 183
    Co-transformed cells grow preferentially as tumors in peritoneal cavities.

    Co-transformed cells grow preferentially as tumors in peritoneal cavities. by Mitchell Hayes (5334887)

    Published 2025
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  4. 184
    Join-point regression analysis of the statistically significant linear decrease in use of antidepressants, antipsychotics, psychostimulants and nootropic drugs, anxiolytics, sedatives and hypnotics during the observed time period.

    Join-point regression analysis of the statistically significant linear decrease in use of antidepressants, antipsychotics, psychostimulants and nootropic drugs, anxiolytics, sedatives and hypnotics during the observed time period. by Jelena Filimonovic (22305307)

    Published 2025
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  5. 185
    Mod is expressed throughout the larval CNS and is primarily nuclear.

    Mod is expressed throughout the larval CNS and is primarily nuclear. by Amalia S. Parra (4173004)

    Published 2024
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  6. 186
    RT-qPCR Primer.

    RT-qPCR Primer. by Dequan Wang (6007007)

    Published 2024
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  7. 187
    Loss of Mod does not affect NB asymmetric cell division.

    Loss of Mod does not affect NB asymmetric cell division. by Amalia S. Parra (4173004)

    Published 2024
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  8. 188
    CCND1 is a target gene of miR-1247-3p.

    CCND1 is a target gene of miR-1247-3p. by Dequan Wang (6007007)

    Published 2024
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  9. 189
    Development of <i>Drosophila</i> Neuroblasts (NB).

    Development of <i>Drosophila</i> Neuroblasts (NB). by Amalia S. Parra (4173004)

    Published 2024
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  10. 190
    miR-1247-3p was downregulated in CRC resistant cells.

    miR-1247-3p was downregulated in CRC resistant cells. by Dequan Wang (6007007)

    Published 2024
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  11. 191
    miR-1247-3p plasmid.

    miR-1247-3p plasmid. by Dequan Wang (6007007)

    Published 2024
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  12. 192
    RNA-sequencing data screening for miR-1247-3p target genes.

    RNA-sequencing data screening for miR-1247-3p target genes. by Dequan Wang (6007007)

    Published 2024
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  13. 193
    Dual luciferase primer.

    Dual luciferase primer. by Dequan Wang (6007007)

    Published 2024
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  14. 194
    Differentially expressed genes following Mod knockdown.

    Differentially expressed genes following Mod knockdown. by Amalia S. Parra (4173004)

    Published 2024
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  15. 195
    shCCND1 plasmid.

    shCCND1 plasmid. by Dequan Wang (6007007)

    Published 2024
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  16. 196
    CCND1 promoted chemoresistance in CRC.

    CCND1 promoted chemoresistance in CRC. by Dequan Wang (6007007)

    Published 2024
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  17. 197
    RNA targets of Mod identified by RiP-Seq.

    RNA targets of Mod identified by RiP-Seq. by Amalia S. Parra (4173004)

    Published 2024
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  18. 198
    NBs in Mod-depleted brains display defects in expression of key cell identity factors.

    NBs in Mod-depleted brains display defects in expression of key cell identity factors. by Amalia S. Parra (4173004)

    Published 2024
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  19. 199
    Loss of Mod causes cell cycle defects.

    Loss of Mod causes cell cycle defects. by Amalia S. Parra (4173004)

    Published 2024
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  20. 200
    Overexpression of miR-1247-3p promoted apoptosis and inhibited resistance in drug-resistant cells.

    Overexpression of miR-1247-3p promoted apoptosis and inhibited resistance in drug-resistant cells. by Dequan Wang (6007007)

    Published 2024
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