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Data_Sheet_1_Porphyromonas gingivalis induces an inflammatory response via the cGAS-STING signaling pathway in a periodontitis mouse model.PDF
Published 2023“…Furthermore, we report that a STING inhibitor (SN-011) significantly decreased inflammatory cytokine production and osteoclast formation in a periodontitis mouse model with P. gingivalis. …”
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Table_1_cGAS/STING and innate brain inflammation following acute high-fat feeding.xlsx
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_1_cGAS/STING and innate brain inflammation following acute high-fat feeding.tif
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_10_cGAS/STING and innate brain inflammation following acute high-fat feeding.tif
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_9_cGAS/STING and innate brain inflammation following acute high-fat feeding.tif
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_3_cGAS/STING and innate brain inflammation following acute high-fat feeding.jpeg
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_2_cGAS/STING and innate brain inflammation following acute high-fat feeding.tif
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_8_cGAS/STING and innate brain inflammation following acute high-fat feeding.tif
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_7_cGAS/STING and innate brain inflammation following acute high-fat feeding.tif
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_4_cGAS/STING and innate brain inflammation following acute high-fat feeding.tif
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_6_cGAS/STING and innate brain inflammation following acute high-fat feeding.tif
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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Image_5_cGAS/STING and innate brain inflammation following acute high-fat feeding.tif
Published 2022“…Central inflammatory changes included microglial activation in a pro-inflammatory environment with cGAS/STING activation. Blocking gap junctions in neuron-microglial co-cultures significantly decreased cGAS/STING activation. …”
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