The table shows the significant and non-significant altered group with different parameters. حسب Ashraf Attia Mahmoud (21262201)

الموضوعات:

Ni causes a decrease in membrane sterols in A549 lung epithelial cells. حسب Amber R. Matha (19681603)

Intracellular retention, but not shedding of NKp65 molecules containing threonine at amino acid position 131 causes decreased surface expression. حسب Julian Leonard Lino Heller (22049674)

IFA significantly attenuated capsule-mediated resistance to the binding of C3b and C5b-9 to bacterial surface. حسب Tingting Wang (123983)

الموضوعات:

ABT199 inhibits ALK ^mutant kinase activity and decreases it phosphorylation. حسب Richard Junior Zapata Dongo (17912565)

الموضوعات:

Loss of Mod causes cell cycle defects. حسب Amalia S. Parra (4173004)

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HAstV-8 (D471S, N512D) spike has decreased or no binding to FcRn compared to wild-type HAstV-8 spike. حسب Catalina Aguilera-Flores (21743843)

Amino acids interactions and binding energy describe that ABT-199 binds to ALK^Mutant. حسب Richard Junior Zapata Dongo (17912565)

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Data Sheet 1_A synonymous mutation of rs1137070 cause the mice Maoa gene transcription and translation to decrease.pdf حسب Kai Xin Li (19696741)

Negative (Prednisone) and basal (ATP) control compounds binding to ALK protein. حسب Richard Junior Zapata Dongo (17912565)

الموضوعات:

Loss of RBM-26 causes mitochondria dysfunction in PLM. حسب Tamjid A. Chowdhury (14149846)

ALK inhibitors (Positive control) and ABT199 (Proposed binding) in complex with the ALK active side. حسب Richard Junior Zapata Dongo (17912565)

الموضوعات:

Mutations in <i>rbm-26</i> cause axon degeneration and axon overlap defects. حسب Tamjid A. Chowdhury (14149846)

Loss of RBM-26 function causes a reduction in the density of mitochondria in the PLM axon. حسب Tamjid A. Chowdhury (14149846)

RBM-26 binds to the <i>mals-1</i> mRNA and negatively regulates its expression. حسب Tamjid A. Chowdhury (14149846)

The binding energy values of ATP, ALK inhibitors and ABT-199 to ALK were obtained from AlphaFold and modeled protein structures. حسب Richard Junior Zapata Dongo (17912565)

الموضوعات:

Overexpression of MALS-1 reduces mitochondria in the PLM axon and causes axon degeneration and axon overlap defects. حسب Tamjid A. Chowdhury (14149846)

"…(F) Decrease in mitochondria density (number of mitochondria per 100 μm) in the PLM axon caused by MALS-1 overexpression. …"

Loss of MALS-1 function suppresses the mitochondrial and axon degeneration phenotypes that are caused by loss of RBM-26 function. حسب Tamjid A. Chowdhury (14149846)

"…(D) Loss of RBM-26 protein expression causes a decrease in mitoribosome expression, which was assayed with the MitoRibo-Tag system consisting of the MRPL-58 mitoribosomal protein tagged at its C-terminus with 3XFLAG (MRPL-58::3XFLAG). …"

Degeneration Saline group presented with significant decreased final weight in relation to other groups. حسب Flavia Gomes Velasque Gama (21175112)

Table_2_A novel anxiety-associated SNP identified in LYNX2 (LYPD1) is associated with decreased protein binding to nicotinic acetylcholine receptors.xlsx حسب Kristin R. Anderson (5478734)