Showing 181 - 200 results of 8,016 for search '(( significant cause decrease ) OR ( significant ((teer decrease) OR (we decrease)) ))', query time: 0.53s Refine Results
  1. 181

    Data for Fig 6D. by Xiaohua Zhang (350570)

    Published 2024
    “…In <i>vivo</i> experiments further showed that PRV UL4 (<sup>132</sup>DVAADAAAEAAAAE<sup>145</sup>) mutated strain (PRV-UL4<sup>mut</sup>) infection did not lead to a significant decrease in viral titers at 12 h. p. i, but it induced lower levels of IL-1β, IL-18, and GSDMD-NT, which led to an alleviated inflammatory infiltration and pathological damage in the lungs and brains, and a lower death rate compared with wild-type PRV strain infection. …”
  2. 182

    Data for Fig 5D. by Xiaohua Zhang (350570)

    Published 2024
    “…In <i>vivo</i> experiments further showed that PRV UL4 (<sup>132</sup>DVAADAAAEAAAAE<sup>145</sup>) mutated strain (PRV-UL4<sup>mut</sup>) infection did not lead to a significant decrease in viral titers at 12 h. p. i, but it induced lower levels of IL-1β, IL-18, and GSDMD-NT, which led to an alleviated inflammatory infiltration and pathological damage in the lungs and brains, and a lower death rate compared with wild-type PRV strain infection. …”
  3. 183

    Data for Figs 8A–8H, 9A–9K, 10A–10I. by Xiaohua Zhang (350570)

    Published 2024
    “…In <i>vivo</i> experiments further showed that PRV UL4 (<sup>132</sup>DVAADAAAEAAAAE<sup>145</sup>) mutated strain (PRV-UL4<sup>mut</sup>) infection did not lead to a significant decrease in viral titers at 12 h. p. i, but it induced lower levels of IL-1β, IL-18, and GSDMD-NT, which led to an alleviated inflammatory infiltration and pathological damage in the lungs and brains, and a lower death rate compared with wild-type PRV strain infection. …”
  4. 184

    Table 1_Dual variants of uncertain significance in a case of hyper-IgM syndrome: implications for diagnosis and management.docx by Nourhen Agrebi (14151222)

    Published 2025
    “…</p>Objective<p>This study aims to elucidate the clinical implications of concurrent AICDA and IKBKB homozygous variants in a pediatric patient diagnosed with hyper-IgM syndrome.</p>Methods<p>We present immunological and genetic analysis of a Tunisian patient with two homozygous variants of uncertain significance (VUSs) in the IKBKB and AICDA genes, suspected of causing hyper-IgM and immune deficiency. …”
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    YOLO11. by Junyan Wang (4738518)

    Published 2025
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    SCI-YOLO11. by Junyan Wang (4738518)

    Published 2025
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    YOLOV8. by Junyan Wang (4738518)

    Published 2025
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    Faster-RCNN. by Junyan Wang (4738518)

    Published 2025
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