Showing 161 - 180 results of 102,642 for search '(( significant changes based ) OR ( significant ((gap decrease) OR (a decrease)) ))', query time: 1.42s Refine Results
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    Using Deep Learning to Fill Data Gaps in Environmental Footprint Accounting by Bu Zhao (9204038)

    Published 2022
    “…For long-term prediction (5 years), the improvements are even more significant where the <i>R</i><sup>2</sup> is improved from 0.5271 to 0.7893 and median average percentage error is decreased from 51.12% to 18.26%. …”
  5. 165

    GTDE decreased the expression of the NLRP3 inflammasome and AGEs in the cerebral cortex. by Hui-Chuan Kuo (12368492)

    Published 2022
    “…Intergroup differences were compared using one-way ANOVA followed by the Tukey’s multiple comparisons test. Statistically significant differences based on comparisons with the control group are denoted by *<i>P</i><0.05, **<i>P</i><0.01, ***<i>P</i><0.001; statistically significant differences based on comparisons with the D-gal group are denoted by <sup>#</sup><i>P</i><0.05, <sup>##</sup><i>P</i><0.01, <sup>###</sup><i>P</i><0.001.…”
  6. 166

    Transcripts with increased translation efficiency during GSC to DGC transition have a significant loss in m6A methylation. by John P. Zepecki (10271291)

    Published 2021
    “…C) Percent composition of top 30% transcripts with most significant change in TE based on m6A status in individual samples (gain/unchanged/loss). …”
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    Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket by Lilia Leisle (11356934)

    Published 2021
    “…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
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    Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket by Lilia Leisle (11356934)

    Published 2021
    “…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
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