Showing 21 - 40 results of 34,817 for search '(( significant decrease cgas ) OR ( significant ((teer decrease) OR (we decrease)) ))', query time: 1.65s Refine Results
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    Changes in TEER and permeability of EA.hy926 cells exposed to EVs or pellets from infected or uninfected macrophages. by Myriam Lucia Velandia-Romero (8259726)

    Published 2020
    “…Interestingly, TEER increased significantly (up to 15 Ω/cm<sup>2</sup>) when EA.hy926 cells were exposed to EVs obtained from infected U937 macrophages (<i>EV (+)</i><sub><i>4G2</i></sub> or <i>P(+))</i> (B). …”
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    Data Sheet 1_Validation of an MPS-based intestinal cell culture model for the evaluation of drug-induced toxicity.docx by Stefanie Hoffmann (3756733)

    Published 2025
    “…Overall, from the 23 tested compounds, 15 showed the expected outcome, i.e., the compound led to a decrease of the TEER for the positive control compounds, or the TEER value remained stable after treatment with non-GI-toxic compounds.…”
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    Data Sheet 3_IFNβ drives ferroptosis through elevating TRIM22 and promotes the cytotoxicity of RSL3.pdf by Huiyue Dong (4808436)

    Published 2025
    “…HT1080 cells, three-dimensional (3D) spheroids, and the xenograft mouse models were treated with IFNβ, RSL3, or IFNβ combination with RSL3 to analyze whether IFNβ enhances RSL3-induced ferroptosis.</p>Results<p>Here, we found that IFNβ could promote intracellular Fe<sup>2+</sup> and lipid peroxidation levels, and decrease GSH levels in tumor cells. …”
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    Data Sheet 1_IFNβ drives ferroptosis through elevating TRIM22 and promotes the cytotoxicity of RSL3.pdf by Huiyue Dong (4808436)

    Published 2025
    “…HT1080 cells, three-dimensional (3D) spheroids, and the xenograft mouse models were treated with IFNβ, RSL3, or IFNβ combination with RSL3 to analyze whether IFNβ enhances RSL3-induced ferroptosis.</p>Results<p>Here, we found that IFNβ could promote intracellular Fe<sup>2+</sup> and lipid peroxidation levels, and decrease GSH levels in tumor cells. …”
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    Data Sheet 2_IFNβ drives ferroptosis through elevating TRIM22 and promotes the cytotoxicity of RSL3.pdf by Huiyue Dong (4808436)

    Published 2025
    “…HT1080 cells, three-dimensional (3D) spheroids, and the xenograft mouse models were treated with IFNβ, RSL3, or IFNβ combination with RSL3 to analyze whether IFNβ enhances RSL3-induced ferroptosis.</p>Results<p>Here, we found that IFNβ could promote intracellular Fe<sup>2+</sup> and lipid peroxidation levels, and decrease GSH levels in tumor cells. …”
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    Data Sheet 2_KAE ameliorates LPS-mediated acute lung injury by inhibiting PANoptosis through the intracellular DNA-cGAS-STING axis.pdf by Yonghu Chen (2040136)

    Published 2025
    “…</p>Results<p>KAE significantly alleviated lipopolysaccharide-induced pulmonary injury by reducing inflammatory cell infiltration, alleviating pulmonary edema, enhancing antioxidant capacity, and decreasing levels of inflammatory cytokines in mouse lung tissues. …”
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    Data Sheet 1_KAE ameliorates LPS-mediated acute lung injury by inhibiting PANoptosis through the intracellular DNA-cGAS-STING axis.zip by Yonghu Chen (2040136)

    Published 2025
    “…</p>Results<p>KAE significantly alleviated lipopolysaccharide-induced pulmonary injury by reducing inflammatory cell infiltration, alleviating pulmonary edema, enhancing antioxidant capacity, and decreasing levels of inflammatory cytokines in mouse lung tissues. …”
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    Manhattan plot that shows the location of genes involved in folate, homocysteine, and transsulfuration pathways for which we observed, from hybrid analyses, a significantly decreased risk of obstructive heart defects when the risk variant was paternally-derived compared to maternally-derived, The National Birth Defects Prevention Study, USA, October 1997–August 2008 births. by Jenil Patel (10271373)

    Published 2021
    “…<p>Manhattan plot that shows the location of genes involved in folate, homocysteine, and transsulfuration pathways for which we observed, from hybrid analyses, a significantly decreased risk of obstructive heart defects when the risk variant was paternally-derived compared to maternally-derived, The National Birth Defects Prevention Study, USA, October 1997–August 2008 births.…”