Loss of Vps34 is associated with decreased eTreg survival but intact activation. حسب Erienne G. Norton (9612079)

Vps34 orchestrates transcriptional and epigenetic remodeling events corresponding to terminal eTreg differentiation during perinatal life. حسب Erienne G. Norton (9612079)

Analysis of Tregs from perinatal mice and mixed BM chimeras for transitional and terminal eTreg states. حسب Erienne G. Norton (9612079)

Targeting of Vps34 reduces B16F10 tumor growth and working model for Vps34-dependent regulation of eTreg transitional heterogeneity and maintenance. حسب Erienne G. Norton (9612079)

Uvrag in Tregs is dispensable for establishment of immune tolerance and eTreg accumulation. حسب Erienne G. Norton (9612079)

Targeting of Vps34 or Atg14, but not Uvrag, in Tregs reduces tumor growth in mouse models. حسب Erienne G. Norton (9612079)

Vps34 orchestrates Treg function in vivo. حسب Erienne G. Norton (9612079)

Atg14 deficiency in Tregs induces a type-I-dominant lymphoproliferative disease and cell-intrinsic reductions in eTregs. حسب Erienne G. Norton (9612079)

Loss of Vps34 reduces Treg suppressive function and is associated with the development of type-I-dominant inflammatory disease. حسب Erienne G. Norton (9612079)

Vps34 supports Treg maintenance in lymphoid and non-lymphoid tissues. حسب Erienne G. Norton (9612079)

Atg14 does not contribute to terminal eTreg generation during perinatal life. حسب Erienne G. Norton (9612079)

Chromatin accessibility analysis of perinatal Vps34-deficient Tregs. حسب Erienne G. Norton (9612079)

Atg14 plays a cell-intrinsic role in orchestrating eTreg survival but not terminal differentiation. حسب Erienne G. Norton (9612079)

DataSheet1_Association between changes in thyroid hormones and incident type 2 diabetes using joint models of longitudinal and time-to-event data: more than a decade follow up in t... حسب Alireza Amirabadizadeh (6399635)

Data Sheet 2_Complement activation drives the phagocytosis of necrotic cell debris and resolution of liver injury.pdf حسب Sofie Vandendriessche (638514)

"…Importantly, C3 deficiency led to a significant accumulation of necrotic debris and impairment of liver recovery in mice, which was attributed to decreased phagocytosis of debris by recruited neutrophils in vivo. …"

Table 1_Complement activation drives the phagocytosis of necrotic cell debris and resolution of liver injury.xlsx حسب Sofie Vandendriessche (638514)

"…Importantly, C3 deficiency led to a significant accumulation of necrotic debris and impairment of liver recovery in mice, which was attributed to decreased phagocytosis of debris by recruited neutrophils in vivo. …"

Video 3_Complement activation drives the phagocytosis of necrotic cell debris and resolution of liver injury.mp4 حسب Sofie Vandendriessche (638514)

"…Importantly, C3 deficiency led to a significant accumulation of necrotic debris and impairment of liver recovery in mice, which was attributed to decreased phagocytosis of debris by recruited neutrophils in vivo. …"

Video 1_Complement activation drives the phagocytosis of necrotic cell debris and resolution of liver injury.mp4 حسب Sofie Vandendriessche (638514)

"…Importantly, C3 deficiency led to a significant accumulation of necrotic debris and impairment of liver recovery in mice, which was attributed to decreased phagocytosis of debris by recruited neutrophils in vivo. …"

Data Sheet 1_Complement activation drives the phagocytosis of necrotic cell debris and resolution of liver injury.docx حسب Sofie Vandendriessche (638514)

"…Importantly, C3 deficiency led to a significant accumulation of necrotic debris and impairment of liver recovery in mice, which was attributed to decreased phagocytosis of debris by recruited neutrophils in vivo. …"

Video 2_Complement activation drives the phagocytosis of necrotic cell debris and resolution of liver injury.mp4 حسب Sofie Vandendriessche (638514)

"…Importantly, C3 deficiency led to a significant accumulation of necrotic debris and impairment of liver recovery in mice, which was attributed to decreased phagocytosis of debris by recruited neutrophils in vivo. …"