Search alternatives:
significant decrease » significant increase (Expand Search), significantly increased (Expand Search)
significant changes » significant challenge (Expand Search)
significant genes » significant gender (Expand Search), significant benefits (Expand Search), significant gap (Expand Search)
significant decrease » significant increase (Expand Search), significantly increased (Expand Search)
significant changes » significant challenge (Expand Search)
significant genes » significant gender (Expand Search), significant benefits (Expand Search), significant gap (Expand Search)
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1161
Ba/F3 EML4-ALK<sup>mutants</sup> exhibit variable dose-response to ABT-199 treatment.
Published 2025Subjects: -
1162
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1163
Molecular docking showed that ABT-199 can fit into active site of ALK<sup>Mutant</sup>.
Published 2025Subjects: -
1164
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1165
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1166
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1167
Amino acids interactions and binding energy describe that ABT-199 binds to ALK<sup>Mutant</sup>.
Published 2025Subjects: -
1168
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1169
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1170
Overexpression of miR-1247-3p promoted apoptosis and inhibited resistance in drug-resistant cells.
Published 2024Subjects: -
1171
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1172
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1173
ALK inhibitors (Positive control) and ABT199 (Proposed binding) in complex with the ALK active side.
Published 2025Subjects: -
1174
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1175
BCL2 antiapoptotic protein exhibits heterogeneous expression in Ba/F3 EML4-ALK<sup>mutants</sup>.
Published 2025Subjects: -
1176
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1177
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1178
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1179
Downregulation of miR-1247-3p inhibited apoptosis and reduced chemosensitivity in cells.
Published 2024Subjects: -
1180
Negative (Prednisone) and basal (ATP) control compounds binding to ALK protein.
Published 2025Subjects: