Loss of FAT1 expression results in increased YAP/TAZ protein levels. by Rui Li (4631)

E3 ligase TRIM56 is up-regulated in response to CVB3 infection. by Yao Wang (102387)

Effect of O-ag modifying glycosyltransferases on complement activation and <i>Salmonella</i> interactions with human serum. by Fermin E. Guerra (6084545)

“…</i> Paratyphi A decrease complement activation as measured by C5a release and (B) IgM binding. …”

Role of YAP/TAZ in FAT1-regulated HNSCC growth. by Rui Li (4631)

O-ag length and composition impacts complement activation by <i>Salmonella</i> nontyphoidal and enteric fever serovars. by Fermin E. Guerra (6084545)

Reduction of L O-ag production and antigenic shift to O2-antigen in by Fermin E. Guerra (6084545)

“…<p><b><i>S.</i> Paratyphi A decreases IgM binding. (</b>A) and (B) Restored <i>S.…”

FAT1 interacts with the E3 ligase MIB2. by Rui Li (4631)

FAT1 and MIB2 control tumor growth <i>in vivo.</i> by Rui Li (4631)

Renal GRK4 knockdown impacted Mas receptor phosphorylation and Its-induced renal function in SHRs. by Lin Chen (54305)

Targeting of Vps34 reduces B16F10 tumor growth and working model for Vps34-dependent regulation of eTreg transitional heterogeneity and maintenance. by Erienne G. Norton (9612079)

The contribution of O-ag length and the Vi capsule to serum resistance is serovar dependent. by Fermin E. Guerra (6084545)

TRIM56 promotes the ubiquitination and proteasomal degradation of CVB3 3D. by Yao Wang (102387)

Analysis of Tregs from perinatal mice and mixed BM chimeras for transitional and terminal eTreg states. by Erienne G. Norton (9612079)

Vps34 orchestrates Treg function in vivo. by Erienne G. Norton (9612079)

Vps34 supports Treg maintenance in lymphoid and non-lymphoid tissues. by Erienne G. Norton (9612079)

“…Two-tailed Student <i>t</i> test (<b>A</b>–<b>D</b>, <b>F</b>–<b>I</b>) or Benjamini–Hochberg test (<b>E</b>); NS, not significant. …”

Chromatin accessibility analysis of perinatal Vps34-deficient Tregs. by Erienne G. Norton (9612079)

“…(<b>A</b>, <b>B</b>). Two-tailed Student <i>t</i> test (<b>A</b>, <b>B</b>, <b>I</b>), Benjamini–Hochberg test (<b>C</b>), Wilcoxon rank-sum test (<b>G</b>), Fisher’s exact tes<i>t</i> (<b>H</b>), or binomial test (<b>J</b>); NS, not significant. …”

Uvrag in Tregs is dispensable for establishment of immune tolerance and eTreg accumulation. by Erienne G. Norton (9612079)

Loss of Vps34 reduces Treg suppressive function and is associated with the development of type-I-dominant inflammatory disease. by Erienne G. Norton (9612079)

Atg14 does not contribute to terminal eTreg generation during perinatal life. by Erienne G. Norton (9612079)

Targeting of Vps34 or Atg14, but not Uvrag, in Tregs reduces tumor growth in mouse models. by Erienne G. Norton (9612079)