Search alternatives:
significant decrease » significant increase (Expand Search), significantly increased (Expand Search)
p decrease » _ decrease (Expand Search), pa decreased (Expand Search), _ decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
significant decrease » significant increase (Expand Search), significantly increased (Expand Search)
p decrease » _ decrease (Expand Search), pa decreased (Expand Search), _ decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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1581
Baseline characteristics of overall population.
Published 2025“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”
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1582
Diagram of study population.
Published 2025“…For every increase in BMI, there was a decrease in the probability of PTB (OR = 0.923, 95% CI 0.915–0.931, P < 0.001). …”
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1583
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1584
Changes in alpha diversity in the weeks before anti-TNF-a treatment.
Published 2024“…The analysis found a significant association across all subjects for Shannon diversity with decreasing diversity in the lead up to the next treatment (lmer model, beta = -0.018, <i>p</i> = 0.036) and no association for observed diversity (<i>p</i>>0.1). …”
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1585
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1586
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1587
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1588
qPCR primer sequences.
Published 2025“…Specifically, <i>ccr2</i> deficiency disrupts the differentiation and response of Th1 cells, which are the primary effector lineage responsible for clearing chlamydia through secretion of interferon-gamma (IFN-γ). As a result, there is a significant decrease in CD3<sup>+</sup>CD4<sup>+</sup>IFN-γ<sup>+</sup> T cells in the lung and spleen, accompanied by reduced levels of IFN-γ protein and mRNA, as well as downregulated mRNA expression of Th1-promoting cytokines (<i>il-12p35</i>, <i>il-12p40</i>) and transcription factors (<i>stat4</i>, <i>T-bet</i>), which play crucial roles in Th1 differentiation. …”
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1589
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1590
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1591
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1592
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1593
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1594
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1595
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1596
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1597
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1598
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1599
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1600