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significant decrease » significant increase (Expand Search), significantly increased (Expand Search)
nucleus increases » cycles increases (Expand Search), numbers increased (Expand Search), number increases (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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Connectivity between the left nucleus accumbens and two clusters.
Published 2025“…After intervention there was a significant decrease in the FR group (mean at V1: 0.249 ± 0.094, mean at V5: 0.130 ± 0.101, difference: −0.119 ± 0.097, p < 0.001) and a significant increase in the SM group (mean at V1: 0.156 ± 0.113, mean at V5: 0.287 ± 0.107, difference: 0.131 ± 0.066, p < 0.001). …”
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Presentation 1_Ceftriaxone-associated dysbiosis decreases voriconazole bioavailability by upregulating intestinal P-glycoprotein expression through activation of the Nrf2-mediated...
Published 2025“…The Nrf2 pathway and its related proteins involved in modifying P-glycoprotein (P-gp) expression were clarified by a series of immunoblot analyses.</p>Key findings<p>The diversity and richness of intestinal bacteria, especially the abundance of gram-negative bacteria, were significantly decreased after ceftriaxone treatment. …”
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Table 1_Ceftriaxone-associated dysbiosis decreases voriconazole bioavailability by upregulating intestinal P-glycoprotein expression through activation of the Nrf2-mediated signall...
Published 2025“…The Nrf2 pathway and its related proteins involved in modifying P-glycoprotein (P-gp) expression were clarified by a series of immunoblot analyses.</p>Key findings<p>The diversity and richness of intestinal bacteria, especially the abundance of gram-negative bacteria, were significantly decreased after ceftriaxone treatment. …”
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Data for Figs 8A–8H, 9A–9K, 10A–10I.
Published 2024“…In <i>vivo</i> experiments further showed that PRV UL4 (<sup>132</sup>DVAADAAAEAAAAE<sup>145</sup>) mutated strain (PRV-UL4<sup>mut</sup>) infection did not lead to a significant decrease in viral titers at 12 h. p. i, but it induced lower levels of IL-1β, IL-18, and GSDMD-NT, which led to an alleviated inflammatory infiltration and pathological damage in the lungs and brains, and a lower death rate compared with wild-type PRV strain infection. …”
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Cis-acting elements in the promoter of PtWRKYs.
Published 2025“…All poplar WRKY proteins are hydrophilic, located in the cell nucleus, and form target relationships with a large number of miRNAs, with their promoter containing a large number of stress defense elements. …”
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The Ka/Ks values of PtWRKY paralogous gene pairs.
Published 2025“…All poplar WRKY proteins are hydrophilic, located in the cell nucleus, and form target relationships with a large number of miRNAs, with their promoter containing a large number of stress defense elements. …”
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RNA-seq data of WRKY genes.
Published 2025“…All poplar WRKY proteins are hydrophilic, located in the cell nucleus, and form target relationships with a large number of miRNAs, with their promoter containing a large number of stress defense elements. …”
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Analysis of PtWRKY genes.
Published 2025“…All poplar WRKY proteins are hydrophilic, located in the cell nucleus, and form target relationships with a large number of miRNAs, with their promoter containing a large number of stress defense elements. …”
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The primer sequences for RT-qPCR.
Published 2025“…All poplar WRKY proteins are hydrophilic, located in the cell nucleus, and form target relationships with a large number of miRNAs, with their promoter containing a large number of stress defense elements. …”
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Data for Fig 6D.
Published 2024“…In <i>vivo</i> experiments further showed that PRV UL4 (<sup>132</sup>DVAADAAAEAAAAE<sup>145</sup>) mutated strain (PRV-UL4<sup>mut</sup>) infection did not lead to a significant decrease in viral titers at 12 h. p. i, but it induced lower levels of IL-1β, IL-18, and GSDMD-NT, which led to an alleviated inflammatory infiltration and pathological damage in the lungs and brains, and a lower death rate compared with wild-type PRV strain infection. …”
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Data for Fig 5D.
Published 2024“…In <i>vivo</i> experiments further showed that PRV UL4 (<sup>132</sup>DVAADAAAEAAAAE<sup>145</sup>) mutated strain (PRV-UL4<sup>mut</sup>) infection did not lead to a significant decrease in viral titers at 12 h. p. i, but it induced lower levels of IL-1β, IL-18, and GSDMD-NT, which led to an alleviated inflammatory infiltration and pathological damage in the lungs and brains, and a lower death rate compared with wild-type PRV strain infection. …”
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