Flow chart of study participants. by Mohamed Achraf Harrabi (22446782)

Subjects:

Participants data. by Mohamed Achraf Harrabi (22446782)

Subjects:

PGAM5 knockdown prevented rTsSPc from disrupting Caco-2 monolayer integrity. by Qi Qi Lu (17721401)

Subjects:

Inhibition of PGAM5 and apoptosis alleviated the rTsSPc-damaged Caco-2 monolayer barrier integrity. by Qi Qi Lu (17721401)

Subjects:

rTsSPc induced Caco-2 cell apoptosis. by Qi Qi Lu (17721401)

Subjects:

Apoptosis inhibitor abolished and restored rTsSPc-disrupted Caco-2 monolayer barrier integrity. by Qi Qi Lu (17721401)

Subjects:

Specific primer sequences of human gut epithelial TJs and apoptosis-related genes for qPCR. by Qi Qi Lu (17721401)

Subjects:

rTsSPc interacted with PGAM5 and increased its protein expression in Caco-2 cells. by Qi Qi Lu (17721401)

Subjects:

Raw data of Figs 1–6 in this study. by Qi Qi Lu (17721401)

Subjects:

Inhibition of PGAM5 and apoptosis prevented <i>T. spiralis</i> larval invasion of Caco-2 monolayer. by Qi Qi Lu (17721401)

Subjects:

The RehaGait® system. by Johanna Rydja (12300797)

“…</p><p>Conclusions</p><p>The patients walked with reduced hip flexion, heel strike angle and toe-off angle, and had shorter strides, decreased velocity, and increased time for swing and single support, compared to HI. …”

Flow chart of included patients and dropouts. by Johanna Rydja (12300797)

“…</p><p>Conclusions</p><p>The patients walked with reduced hip flexion, heel strike angle and toe-off angle, and had shorter strides, decreased velocity, and increased time for swing and single support, compared to HI. …”

Dataset of the analyzed data. by Johanna Rydja (12300797)

“…</p><p>Conclusions</p><p>The patients walked with reduced hip flexion, heel strike angle and toe-off angle, and had shorter strides, decreased velocity, and increased time for swing and single support, compared to HI. …”

Average wellness for matchday, MD by Ivan Baptista (5278874)

“…<div><p>Background</p><p>Exposure to match loads significantly affects physiological and psychological indicators and, consequently, players’ wellness. …”

Atg14 deficiency in Tregs induces a type-I-dominant lymphoproliferative disease and cell-intrinsic reductions in eTregs. by Erienne G. Norton (9612079)

Loss of Vps34 reduces Treg suppressive function and is associated with the development of type-I-dominant inflammatory disease. by Erienne G. Norton (9612079)

Vps34 supports Treg maintenance in lymphoid and non-lymphoid tissues. by Erienne G. Norton (9612079)

“…Two-tailed Student <i>t</i> test (<b>A</b>–<b>D</b>, <b>F</b>–<b>I</b>) or Benjamini–Hochberg test (<b>E</b>); NS, not significant. Data are compiled from 15 (<b>A</b>, <b>F</b>), 12 (<b>B</b>), 5 (<b>C</b>, <b>G</b>, <b>I</b>), 6 (<b>D</b>, lung and liver; <b>H</b>), or 2 (<b>D</b>, colon LP) independent experiments. …”

Flowchart of the study population. by Emma Hertting (21568077)

“…<div><p>Objective</p><p>To investigate the effect of antenatal identification of small for gestational age (SGA) fetuses on perinatal and childhood outcomes, separately analyzing early- and late-onset fetal growth restriction (FGR).…”

Atg14 does not contribute to terminal eTreg generation during perinatal life. by Erienne G. Norton (9612079)

Chromatin accessibility analysis of perinatal Vps34-deficient Tregs. by Erienne G. Norton (9612079)

“…Transcription factors with predicted decreased activity, including AP-1-related transcription factors, are labeled. …”