Loss of Mod does not affect NB asymmetric cell division. by Amalia S. Parra (4173004)

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CCND1 is a target gene of miR-1247-3p. by Dequan Wang (6007007)

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Development of <i>Drosophila</i> Neuroblasts (NB). by Amalia S. Parra (4173004)

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miR-1247-3p was downregulated in CRC resistant cells. by Dequan Wang (6007007)

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RNA-sequencing data screening for miR-1247-3p target genes. by Dequan Wang (6007007)

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Differentially expressed genes following Mod knockdown. by Amalia S. Parra (4173004)

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CCND1 promoted chemoresistance in CRC. by Dequan Wang (6007007)

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RNA targets of Mod identified by RiP-Seq. by Amalia S. Parra (4173004)

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NBs in Mod-depleted brains display defects in expression of key cell identity factors. by Amalia S. Parra (4173004)

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Loss of Mod causes cell cycle defects. by Amalia S. Parra (4173004)

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Overexpression of miR-1247-3p promoted apoptosis and inhibited resistance in drug-resistant cells. by Dequan Wang (6007007)

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Effect of miR-1247-3p on survival in colorectal cancer patients. by Dequan Wang (6007007)

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Mod antibody and RNAi validations. by Amalia S. Parra (4173004)

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Mod is required to maintain nucleolar architecture. by Amalia S. Parra (4173004)

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Differential gene expression analysis following Mod knockdown. by Amalia S. Parra (4173004)

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Mod loss is associated with a reduction of central brain NBs. by Amalia S. Parra (4173004)

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RNA targets and biological process GO terms associated with Mod RiP-Seq analysis. by Amalia S. Parra (4173004)

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Downregulation of miR-1247-3p inhibited apoptosis and reduced chemosensitivity in cells. by Dequan Wang (6007007)

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Mod localizes both within and outside the nucleolus in NBs. by Amalia S. Parra (4173004)

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miR-1247-3p was downregulated in tissues of patients with CRC and was associated with prognosis and drug resistance. by Dequan Wang (6007007)

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