Table 1_Exploring the therapeutic potential of “Zhi-Zhen” formula for oxaliplatin resistance in colorectal cancer: an integrated study combining UPLC-QTOF-MS/MS, bioinformatics, ne... by Yongjing Li (3821944)

“…Background<p>Chemoresistance is a critical factor compromising the survival of patients with colorectal cancer (CRC). …”

Table 1_The RNA-binding protein ProQ directly binds and regulates virulence genes in enterohemorrhagic Escherichia coli O157:H7.docx by Ye Qian (7400186)

“…However, the impact of ProQ in many pathogenic bacteria remains poorly understood. In this study, we investigated the role of ProQ in enterohaemorrhagic Escherichia coli (EHEC), a non-invasive intestinal pathogen. …”

Physicochemical property and archaea community characteristics in casing soil of cultivating Stropharia rugosoannulata by Songhao Tian (22609826)

“…Organic nitrogen content was for was significantly higher in A4 stage at 0.276 g/kg. organic content was significantly higher in A1 and A4 stages (0.401 g/kg, 0.397 g/kg). …”

Image 2_Impaired K48-polyubiquitination downmodulates mouse norovirus propagation.tiff by Emmrich Wakeford (21249104)

“…</p>Results<p>Upon infection of these cells with MNoV_S99, we unexpectedly observed that only cells expressing the YFP-Ubiquitin_K48R protein showed a significantly impaired expression of several viral markers: NS5, NS7, VP1 and the replication intermediate dsRNA. …”

Image 1_Impaired K48-polyubiquitination downmodulates mouse norovirus propagation.tiff by Emmrich Wakeford (21249104)

“…</p>Results<p>Upon infection of these cells with MNoV_S99, we unexpectedly observed that only cells expressing the YFP-Ubiquitin_K48R protein showed a significantly impaired expression of several viral markers: NS5, NS7, VP1 and the replication intermediate dsRNA. …”

Data Sheet 1_Influence of specific tobacco endophytic Bacillus on tobacco leaf quality enhancement during fermentation.pdf by Jinbin Wei (492099)

“…Chemical analysis revealed that fermentation with B. halotolerans NS36 significantly decreased the content of irritant compounds such as lignin, cellulose, starch, and pectin. …”

Roxadustat alleviates cisplatin-induced acute kidney injury by regulating fatty acid oxidation and mitochondrial function by Xialin Li (20449207)

“…</p> <p>We investigated the effects of roxadustat (10 mg/kg) in a cisplatin-induced AKI model in mice. …”

Data and code for "Sympathetic vasomotion as an early marker of hemorrhage." by Peter Pellegrino (20875475)

“…The sympathetic nervous system is the fastest physiological hemodynamic compensatory mechanism, and we have developed a novel measure of sympathetic vascular control called sympathetic vasomotion which could serve as an early marker of hemorrhage. …”

Supplementary Figures_Fainshtein.pdf by Kfir Sharabi (21415808)

“…Furthermore, we show that depleting <i>Hdac1</i> in mouse primary hepatocytes and liver tissue reduces glucose production, consistent with decreased PGC-1α activity. …”

Table 8_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 4_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 7_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 10_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 2_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 11_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 1_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 9_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 5_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 3_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”

Table 6_CX3CR1 upregulation modulates microglial activation and preserves synapses in the hippocampus and frontal cortex of middle-aged mice.xlsx by Jinfeng Liu (32678)

“…Further proteomic analysis indicated that CX3CR1 deficiency affected protein levels in GO pathways such as “glutamatergic synapse” and “RNA splicing.” Additionally, we observed a reduction in synaptophysin-positive synapse density in both the hippocampus and frontal cortex of CX3CR1-deficient mice.…”