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significant decrease » significant increase (Expand Search), significantly increased (Expand Search)
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significant decrease » significant increase (Expand Search), significantly increased (Expand Search)
significant a » significant _ (Expand Search), significant i (Expand Search), significant gap (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency
Published 2024“…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …”
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4336
Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency
Published 2024“…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …”
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4337
Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency
Published 2024“…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …”
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4338
Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency
Published 2024“…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …”
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4339
Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency
Published 2024“…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …”
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4340
Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency
Published 2024“…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …”