Phagocytosis of fluorescent latex beads (2 μm) by uninfected and infected PEMs, BMMs, and THP-1 (C) in static culture system. حسب Alaa Riezk (7037297)

الموضوعات:

Macropinocytosis of pHrodo Red dextran by uninfected and infected PEMs, BMMs and THP-1) in static culture system. حسب Alaa Riezk (7037297)

الموضوعات:

Phagocytosis of fluorescent latex beads (2 μm) by infected PEMs, BMMs and THP-1 in the three culture systems (static, slow flow rate 1.45 x 10− − 9 m/s and fast flow rate 1.23 x 10... حسب Alaa Riezk (7037297)

الموضوعات:

Macropinocytosis of pHrodo Red dextran by infected PEMs, BMMs and THP-1 at the three culture systems (static, slow flow rate 1.45 x 10− − 9 m/s and fast flow rate 1.23 x 10− − 7 m/... حسب Alaa Riezk (7037297)

الموضوعات:

<i>In vitro</i> activity of chitosan solution and nanoparticles against <i>L. major</i> amastigotes in RPMI-1640 (pH = 6.5) at different flow rates. حسب Alaa Riezk (7037297)

الموضوعات:

Dose-response curve of the activity of chitosan solution (A), blank chitosan-TPP nanoparticles (B), AmB loaded chitosan-TPP nanoparticles (C) and AmB solution (pure) (D) against L. حسب Alaa Riezk (7037297)

الموضوعات:

RBM-26 binds to the <i>mals-1</i> mRNA and negatively regulates its expression. حسب Tamjid A. Chowdhury (14149846)

Loss of MALS-1 function suppresses the mitochondrial and axon degeneration phenotypes that are caused by loss of RBM-26 function. حسب Tamjid A. Chowdhury (14149846)

"…Asterisks indicate statistically significant difference relative to wild type, Student’s <i>t</i> test (*<i>p</i> < 0.05). …"

Expression of RBM-26 is disrupted by P80L and L13V missense mutations. حسب Tamjid A. Chowdhury (14149846)

WDR12 associates with Aurora B. حسب Zachary J. Hough (20840430)

ROS reduces the expression of Akt3, WDR12 and Aurora B leading to micronuclei. حسب Zachary J. Hough (20840430)

Immunofluorescence staining of p65 NF-κB in Poly (I:C) treated and untreated PK-15 cells at 2, 8, 24 and 48 hours post treatment. حسب Huan W. Yang (19746645)

Analysis of Tregs from perinatal mice and mixed BM chimeras for transitional and terminal eTreg states. حسب Erienne G. Norton (9612079)

Targeting of Vps34 reduces B16F10 tumor growth and working model for Vps34-dependent regulation of eTreg transitional heterogeneity and maintenance. حسب Erienne G. Norton (9612079)

Uvrag in Tregs is dispensable for establishment of immune tolerance and eTreg accumulation. حسب Erienne G. Norton (9612079)

Targeting of Vps34 or Atg14, but not Uvrag, in Tregs reduces tumor growth in mouse models. حسب Erienne G. Norton (9612079)

Vps34 orchestrates Treg function in vivo. حسب Erienne G. Norton (9612079)

Atg14 deficiency in Tregs induces a type-I-dominant lymphoproliferative disease and cell-intrinsic reductions in eTregs. حسب Erienne G. Norton (9612079)

Loss of Vps34 reduces Treg suppressive function and is associated with the development of type-I-dominant inflammatory disease. حسب Erienne G. Norton (9612079)

Vps34 supports Treg maintenance in lymphoid and non-lymphoid tissues. حسب Erienne G. Norton (9612079)