Baseline and Post-Treatment Myocardial Perfusion Defects Expressed as a Percentage of the Total Left Ventricular Surface Area in Each Experimental Group. by Káryta Suely Macêdo Martins (22404155)

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Baseline and post-treatment results of quantitative electrocardiographic variables in each study group. by Káryta Suely Macêdo Martins (22404155)

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CONSORT diagram illustrating patient flow during the study. by Káryta Suely Macêdo Martins (22404155)

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Baseline and Post-Treatment Results of Echocardiographic Variables in Each Experimental Group. by Káryta Suely Macêdo Martins (22404155)

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Illustrative images of the processing of a myocardial perfusion study in a patient with Chagas disease. by Káryta Suely Macêdo Martins (22404155)

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Clinical and Demographic Characteristics of the Study Population. by Káryta Suely Macêdo Martins (22404155)

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Illustrative images of the quantitative processing of polar maps from a myocardial perfusion study in one of the investigated patients. by Káryta Suely Macêdo Martins (22404155)

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Baseline and Post-Treatment Serum Cytokine Levels in Each Investigational Group. by Káryta Suely Macêdo Martins (22404155)

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Baseline and Post-Treatment Serum Levels of NT-proBNP in Each Investigational Group. by Káryta Suely Macêdo Martins (22404155)

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Variables obtained from the SF-36 quality of life questionnaire in each experimental group. by Káryta Suely Macêdo Martins (22404155)

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Line graph showing the values of myocardial perfusion defects in the study groups. p = ANOVA Mixed Models. by Káryta Suely Macêdo Martins (22404155)

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All data points from Fig 2. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 5. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 8. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 3. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 1. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 4. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 9. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 7. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 6. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”