Loss of MALS-1 function suppresses the mitochondrial and axon degeneration phenotypes that are caused by loss of RBM-26 function. by Tamjid A. Chowdhury (14149846)

“…Asterisks indicate statistically significant difference relative to wild type, Student’s <i>t</i> test (*<i>p</i> < 0.05). …”

The growth of co-transformed cells in the peritoneal cavity alters viral gene expression to mirror that of PEL cell lines. by Mitchell Hayes (5334887)

Overexpression of MALS-1 reduces mitochondria in the PLM axon and causes axon degeneration and axon overlap defects. by Tamjid A. Chowdhury (14149846)

“…Asterisks in panels D and E indicate statistically significant difference relative to wild type, Z-test for proportions (***<i>p</i> < 0.0001). …”

The growth of co-transformed cells in the peritoneal cavity alters their cellular gene expression to mirror that of PEL biopsies. by Mitchell Hayes (5334887)

Co-infected cells express both KSHV and EBV genes <i>in vitro</i> necessary for their survival and growth. by Mitchell Hayes (5334887)

Loss of RBM-26 causes mitochondria dysfunction in PLM. by Tamjid A. Chowdhury (14149846)

RBM-26 is expressed in the PLM and functions within neurons to protect against axon degeneration and prevent axon overlap defects. by Tamjid A. Chowdhury (14149846)

Mutations in <i>rbm-26</i> cause axon degeneration and axon overlap defects. by Tamjid A. Chowdhury (14149846)

Summary of Biomarker Expression in Benign Samples. The STT and LTT groups exhibit significant changes in biomarker expression compared to the nontreatment group, measured on a two-... by Jinho Lee (587173)

Subjects:

Proportion of stillbirths in Stockholm among twin and singleton pregnancies. by Matilda Varli Hjärne (22766125)

Loss of RBM-26 function causes a reduction in the density of mitochondria in the PLM axon. by Tamjid A. Chowdhury (14149846)

Primers for RT-qPCR. by Jiaoyang Li (2862269)

“…Recombinant PRRSV lacking this motif displayed reduced virulence and decreased SH3KBP1 degradation. This study advances our understanding of how PRRSV interferes with the host immune response and offers valuable insights for developing novel attenuated vaccines against PRRSV.…”

HBV RNaseH inhibitors effects on capsid accumulation occur independent of HBV DNA synthesis. by Daniel P. Bradley (10306893)

Co-transformed cells grow preferentially as tumors in peritoneal cavities. by Mitchell Hayes (5334887)

Expression of RBM-26 is disrupted by P80L and L13V missense mutations. by Tamjid A. Chowdhury (14149846)

Effects of 1466 on capsid and pgRNA encapsidation compared to known CAM-A and CAM-E inhibitors. by Daniel P. Bradley (10306893)

Screening HBV RNaseH inhibitors in HepDES19 cell for effects on capsids and core protein. by Daniel P. Bradley (10306893)

1466 inhibits capsid accumulation independent of HBV replication. by Daniel P. Bradley (10306893)

Changes in the genotypes of <i>NLRP1</i>, <i>NLRP3</i>, and <i>NLRP4</i>. by Hao Deng (409186)

Subjects:

Change in the genotypes of <i>ATM</i>, <i>ERCC1</i>, and <i>XRCC1</i>. by Hao Deng (409186)

Subjects: