Showing 1,321 - 1,340 results of 1,562 for search '(( significant decrease decrease ) OR ( significantly linked decrease ))~', query time: 0.23s Refine Results
  1. 1321

    Table 3_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.xlsx by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  2. 1322

    Table 8_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.xlsx by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  3. 1323

    Table 2_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.xlsx by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  4. 1324

    Table 1_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.xlsx by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  5. 1325

    Image 2_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.tif by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  6. 1326

    Table 6_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.xlsx by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  7. 1327

    Table 5_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.xlsx by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  8. 1328

    Image 5_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.tif by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  9. 1329

    Image 1_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.tiff by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  10. 1330

    Table 7_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.xlsx by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  11. 1331

    Table 4_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.xlsx by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  12. 1332

    Image 6_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.tif by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  13. 1333

    Image 4_Cellular interactions and Ion channel signatures in atrial fibrillation remodeling: insights from single-cell analysis and machine learning.tif by Bin He (75597)

    Published 2025
    “…These subtypes exhibited significantly different distributions between AF and SR. …”
  14. 1334

    Table3_Aconitate decarboxylase 1 mediates the acute airway inflammatory response to environmental exposures.docx by Aaron D. Schwab (18289974)

    Published 2024
    “…Aconitate decarboxylase 1 (Acod1) was one of the most upregulated genes following LPS (vs. saline) exposure of murine whole lungs with transcriptomic profiling of sorted lung monocyte/macrophage subpopulations also highlighting its significance. Given monocyte/macrophage activation can be tightly linked to metabolism, the objective of these studies was to determine the role of the immunometabolic regulator ACOD1 in environmental exposure-induced lung inflammation.…”
  15. 1335

    Image1_Aconitate decarboxylase 1 mediates the acute airway inflammatory response to environmental exposures.jpeg by Aaron D. Schwab (18289974)

    Published 2024
    “…Aconitate decarboxylase 1 (Acod1) was one of the most upregulated genes following LPS (vs. saline) exposure of murine whole lungs with transcriptomic profiling of sorted lung monocyte/macrophage subpopulations also highlighting its significance. Given monocyte/macrophage activation can be tightly linked to metabolism, the objective of these studies was to determine the role of the immunometabolic regulator ACOD1 in environmental exposure-induced lung inflammation.…”
  16. 1336

    Table1_Aconitate decarboxylase 1 mediates the acute airway inflammatory response to environmental exposures.docx by Aaron D. Schwab (18289974)

    Published 2024
    “…Aconitate decarboxylase 1 (Acod1) was one of the most upregulated genes following LPS (vs. saline) exposure of murine whole lungs with transcriptomic profiling of sorted lung monocyte/macrophage subpopulations also highlighting its significance. Given monocyte/macrophage activation can be tightly linked to metabolism, the objective of these studies was to determine the role of the immunometabolic regulator ACOD1 in environmental exposure-induced lung inflammation.…”
  17. 1337

    Table2_Aconitate decarboxylase 1 mediates the acute airway inflammatory response to environmental exposures.docx by Aaron D. Schwab (18289974)

    Published 2024
    “…Aconitate decarboxylase 1 (Acod1) was one of the most upregulated genes following LPS (vs. saline) exposure of murine whole lungs with transcriptomic profiling of sorted lung monocyte/macrophage subpopulations also highlighting its significance. Given monocyte/macrophage activation can be tightly linked to metabolism, the objective of these studies was to determine the role of the immunometabolic regulator ACOD1 in environmental exposure-induced lung inflammation.…”
  18. 1338

    Image3_Aconitate decarboxylase 1 mediates the acute airway inflammatory response to environmental exposures.jpeg by Aaron D. Schwab (18289974)

    Published 2024
    “…Aconitate decarboxylase 1 (Acod1) was one of the most upregulated genes following LPS (vs. saline) exposure of murine whole lungs with transcriptomic profiling of sorted lung monocyte/macrophage subpopulations also highlighting its significance. Given monocyte/macrophage activation can be tightly linked to metabolism, the objective of these studies was to determine the role of the immunometabolic regulator ACOD1 in environmental exposure-induced lung inflammation.…”
  19. 1339

    Image2_Aconitate decarboxylase 1 mediates the acute airway inflammatory response to environmental exposures.jpeg by Aaron D. Schwab (18289974)

    Published 2024
    “…Aconitate decarboxylase 1 (Acod1) was one of the most upregulated genes following LPS (vs. saline) exposure of murine whole lungs with transcriptomic profiling of sorted lung monocyte/macrophage subpopulations also highlighting its significance. Given monocyte/macrophage activation can be tightly linked to metabolism, the objective of these studies was to determine the role of the immunometabolic regulator ACOD1 in environmental exposure-induced lung inflammation.…”
  20. 1340

    Table2_The short-chain fatty acid receptors Gpr41/43 regulate bone mass by promoting adipogenic differentiation of mesenchymal stem cells.xlsx by Friederike Behler-Janbeck (3384488)

    Published 2024
    “…Recently, it has been shown that bone turnover decreases shortly after food consumption. This process has been linked to the fermentation of non-digestible food ingredients such as inulin by gut microbes, which results in the production of the short-chain fatty acids (SCFAs) acetate, propionate and butyrate. …”