Vps34 orchestrates transcriptional and epigenetic remodeling events corresponding to terminal eTreg differentiation during perinatal life. by Erienne G. Norton (9612079)

“…Transcription factors with predicted decreased activity are labeled, including AP-1-related, PPARγ, and NFκB-related transcription factors. …”

Chromatin accessibility analysis of perinatal Vps34-deficient Tregs. by Erienne G. Norton (9612079)

“…Transcription factors with predicted decreased activity, including AP-1-related transcription factors, are labeled. …”

Related to Fig 2. by Mohammad Nafees Ansari (22232505)

Quantification of fasting glucose, random glucose, and insulin levels. by Jessica C. M. Hui (20370522)

Measurements of body mass index (BMI) and adipose deposits. by Jessica C. M. Hui (20370522)

pI7L suppresses the IFN-<i>γ</i>-triggered JAK-STAT signaling pathway. by Meilin Li (1832905)

Bone-related serum chemistry endpoints: Aquamin (180-day treatment group) versus placebo. by Muhammad N. Aslam (6598001)

“…A composite score was generated by combining the three individual endpoint scores. Statistical significance between group (composite) values was determined by unpaired t-test; asterisk (*) indicates p < 0.05. …”

Uvrag in Tregs is dispensable for establishment of immune tolerance and eTreg accumulation. by Erienne G. Norton (9612079)

Atg14 deficiency in Tregs induces a type-I-dominant lymphoproliferative disease and cell-intrinsic reductions in eTregs. by Erienne G. Norton (9612079)

Vps34 orchestrates Treg function in vivo. by Erienne G. Norton (9612079)

Loss of Vps34 reduces Treg suppressive function and is associated with the development of type-I-dominant inflammatory disease. by Erienne G. Norton (9612079)

Atg14 plays a cell-intrinsic role in orchestrating eTreg survival but not terminal differentiation. by Erienne G. Norton (9612079)

“…Jun/Fos transcription factors with predicted decreased activity in Atg14-deficient versus control eTregs are labeled. …”

Vps34 supports Treg maintenance in lymphoid and non-lymphoid tissues. by Erienne G. Norton (9612079)

“…Two-tailed Student <i>t</i> test (<b>A</b>–<b>D</b>, <b>F</b>–<b>I</b>) or Benjamini–Hochberg test (<b>E</b>); NS, not significant. Data are compiled from 15 (<b>A</b>, <b>F</b>), 12 (<b>B</b>), 5 (<b>C</b>, <b>G</b>, <b>I</b>), 6 (<b>D</b>, lung and liver; <b>H</b>), or 2 (<b>D</b>, colon LP) independent experiments. …”

Atg14 does not contribute to terminal eTreg generation during perinatal life. by Erienne G. Norton (9612079)

<i>S</i>. Paratyphi A has deficient L O-ag production due to R98C mutation in WzzB. by Fermin E. Guerra (6084545)

Related to Fig 1. by Mohammad Nafees Ansari (22232505)

Related to Fig 3. by Mohammad Nafees Ansari (22232505)

Prevalence of CSF biomarkers across age. by Sarah J. Neal (20721301)

WGCNA as well as the co-expression network. by Hao Wang (39217)

Summary of related works. by Haichao Li (225035)