Showing 101 - 120 results of 77,601 for search '(( significant graph based ) OR ( significant ((gap decrease) OR (a decrease)) ))', query time: 3.49s Refine Results
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    Overview of long-range interaction prediction and graph-based variant interpretation. by Brittany Baur (1804336)

    Published 2023
    “…</b> SNPs and genes are connected in the SNP-gene network via long-range interactions with the SNPs for a given phenotype. Genes are scored based on the average significance of interactions with SNPs <b>D.…”
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    Optogenetic stimulation of the hippocampal CA1 astrocytes does not significantly affect the expression of AMPA receptors or VGLUT2. by Jae-Hong Kim (124622)

    Published 2024
    “…Quantification of the band intensities is presented in the adjacent graphs. Results are expressed as mean ± SD (<i>n</i> = 4). n.s., not significant (one-way ANOVA). …”
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    LCN2 treatment does not significantly alter the membrane expression of AMPA receptors or VGLUT2 in the hippocampal neurons. by Jae-Hong Kim (124622)

    Published 2024
    “…Quantification of GluR1, GluR2, Homer, or VGLUT2 in PNS was based on normalization against the internal control β-actin, and that of GLUR1, GLUR2, Homer, or VGLUT2 in SPM was normalized against the internal control Ponceau S (<a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.3002687#pbio.3002687.s013" target="_blank">S13C Fig</a>) and represented as graphs for the blots. …”
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    Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket by Lilia Leisle (11356934)

    Published 2021
    “…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
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    Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket by Lilia Leisle (11356934)

    Published 2021
    “…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
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