Atg14 does not contribute to terminal eTreg generation during perinatal life. حسب Erienne G. Norton (9612079)

Chromatin accessibility analysis of perinatal Vps34-deficient Tregs. حسب Erienne G. Norton (9612079)

"…Relative (normalized to average control in each experiment) fold-change comparisons between genotypes in each cell type are shown in bold (<b>A</b>, <b>B</b>). Data are shown as mean ±  s.e.m. (<b>A</b>, <b>B</b>). Two-tailed Student <i>t</i> test (<b>A</b>, <b>B</b>, <b>I</b>), Benjamini–Hochberg test (<b>C</b>), Wilcoxon rank-sum test (<b>G</b>), Fisher’s exact tes<i>t</i> (<b>H</b>), or binomial test (<b>J</b>); NS, not significant. …"

Atg14 plays a cell-intrinsic role in orchestrating eTreg survival but not terminal differentiation. حسب Erienne G. Norton (9612079)

"…Jun/Fos transcription factors with predicted decreased activity in Atg14-deficient versus control eTregs are labeled. …"

Analysis of Tregs from perinatal mice and mixed BM chimeras for transitional and terminal eTreg states. حسب Erienne G. Norton (9612079)

Vps34 orchestrates transcriptional and epigenetic remodeling events corresponding to terminal eTreg differentiation during perinatal life. حسب Erienne G. Norton (9612079)

"…See also <a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.3003074#pbio.3003074.s017" target="_blank">S9 Table</a>. Data are shown as mean ± s.e.m. (<b>D</b>). Wilcoxon rank-sum test (<b>A</b>, <b>B</b>; <b>J</b>, right), Benjamini–Hochbert test (<b>C</b>), two-tailed Student <i>t</i> test (<b>D</b>, <b>I</b>), Fisher’s exact test (<b>F</b>–<b>H</b>), or bionomial test (<b>J</b>, left); NS, not significant. …"

Uvrag in Tregs is dispensable for establishment of immune tolerance and eTreg accumulation. حسب Erienne G. Norton (9612079)

Targeting of Vps34 or Atg14, but not Uvrag, in Tregs reduces tumor growth in mouse models. حسب Erienne G. Norton (9612079)

Atg14 deficiency in Tregs induces a type-I-dominant lymphoproliferative disease and cell-intrinsic reductions in eTregs. حسب Erienne G. Norton (9612079)

Expression of 20 CrBV genes in different tissues of FAW following parasitism by <i>Cotesia ruficrus.</i> حسب Xian Li (201841)

Expression of 20 CrBV genes in FAW at different time points following parasitism by <i>Cotesia ruficrus.</i> حسب Xian Li (201841)

Expression of 20 CrBV genes in FAW following <i>CrBV3–31</i> interference. حسب Xian Li (201841)

Expression of 20 CrBV genes in FAW following <i>CrBV13–7</i> interference. حسب Xian Li (201841)

Expression of 20 CrBV genes in FAW following <i>CrBV11–6</i> interference. حسب Xian Li (201841)

Effect of parasitism and PDV injection on triglyceride concentration and lipid metabolism-related gene expression in FAW larvae. حسب Xian Li (201841)

Effect of parasitism and PDV injection on lipid droplet size in the fat body of FAW larvae. حسب Xian Li (201841)

ASFV-ΔI7L induces higher immune responses than does ASFV-WT in pigs. حسب Meilin Li (1832905)

Differential expression profiling in the ASFV-ΔI7L-infected primary porcine alveolar macrophages (PAMs) by RNA sequencing analysis. حسب Meilin Li (1832905)

pI7L suppresses the IFN-γ-triggered JAK-STAT signaling pathway. حسب Meilin Li (1832905)

The <i>I7L</i> gene is involved in ASFV pathogenicity in pigs. حسب Meilin Li (1832905)

Effect of <i>CrBV3-31</i> interference on lipid metabolism in FAW larvae. حسب Xian Li (201841)