Loss of Mod does not affect NB asymmetric cell division. by Amalia S. Parra (4173004)

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Mod localizes both within and outside the nucleolus in NBs. by Amalia S. Parra (4173004)

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Mod is expressed throughout the larval CNS and is primarily nuclear. by Amalia S. Parra (4173004)

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RT-qPCR Primer. by Dequan Wang (6007007)

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miR-1247-3p plasmid. by Dequan Wang (6007007)

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Dual luciferase primer. by Dequan Wang (6007007)

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shCCND1 plasmid. by Dequan Wang (6007007)

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S1 Raw images - by Dequan Wang (6007007)

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Introduction to the dataset. by Dequan Wang (6007007)

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Loss of Mod causes cell cycle defects. by Amalia S. Parra (4173004)

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Differential gene expression analysis following Mod knockdown. by Amalia S. Parra (4173004)

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NBs in Mod-depleted brains display defects in expression of key cell identity factors. by Amalia S. Parra (4173004)

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Mod antibody and RNAi validations. by Amalia S. Parra (4173004)

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RNA targets and biological process GO terms associated with Mod RiP-Seq analysis. by Amalia S. Parra (4173004)

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Differentially expressed genes following Mod knockdown. by Amalia S. Parra (4173004)

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CCND1 promoted chemoresistance in CRC. by Dequan Wang (6007007)

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miR-1247-3p was downregulated in CRC resistant cells. by Dequan Wang (6007007)

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Downregulation of miR-1247-3p inhibited apoptosis and reduced chemosensitivity in cells. by Dequan Wang (6007007)

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miR-1247-3p was downregulated in tissues of patients with CRC and was associated with prognosis and drug resistance. by Dequan Wang (6007007)

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Overexpression of miR-1247-3p promoted apoptosis and inhibited resistance in drug-resistant cells. by Dequan Wang (6007007)

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