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significant source » significant burden (Expand Search), significant cause (Expand Search)
step decrease » sizes decrease (Expand Search), teer decrease (Expand Search), we decrease (Expand Search)
source based » resource based (Expand Search), surface based (Expand Search), sources used (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
significant source » significant burden (Expand Search), significant cause (Expand Search)
step decrease » sizes decrease (Expand Search), teer decrease (Expand Search), we decrease (Expand Search)
source based » resource based (Expand Search), surface based (Expand Search), sources used (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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Optogenetic stimulation of the hippocampal CA1 astrocytes does not significantly affect the expression of AMPA receptors or VGLUT2.
Published 2024“…Results are expressed as mean ± SD (<i>n</i> = 4). n.s., not significant (one-way ANOVA). Source data can be found in <a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.3002687#pbio.3002687.s025" target="_blank">S1 Data</a>.…”
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LCN2 treatment does not significantly alter the membrane expression of AMPA receptors or VGLUT2 in the hippocampal neurons.
Published 2024“…*<i>p</i> < 0.05 between the indicated groups; n.s., not significant (one-way ANOVA). (<b>C</b>) Western blot analysis of GLUR1 or GLUR2 present in SPM obtained from the hippocampal neurons after glycine treatment. …”
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Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket
Published 2021“…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
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Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket
Published 2021“…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
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