Validation of model predictions of growth during infusion of hypertrophic agonists with and without receptor blockers. حسب Johane H. Bracamonte (12883844)

"…By contrast, the model prediction of nearly equal likelihood that ERA increases or decreases NE-induced growth appears to contradict a significant reported decrease in the only available experimental study [<a href="http://www.ploscompbiol.org/article/info:doi/10.1371/journal.pcbi.1012390#pcbi.1012390.ref121" target="_blank">121</a>]. …"

T3SS induction slows growth, but cells can support high levels of S10 expression. حسب Justin Greene (306625)

Droplet-based microfluidics can be used to model clustered bacterial growth. حسب Justin Greene (306625)

Predicted chronic activation of the cardiomyocyte signaling network during VO. حسب Johane H. Bracamonte (12883844)

Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency حسب Caiping Zhang (6745976)

"…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …"

Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency حسب Caiping Zhang (6745976)

"…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …"

Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency حسب Caiping Zhang (6745976)

"…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …"

Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency حسب Caiping Zhang (6745976)

"…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …"

Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency حسب Caiping Zhang (6745976)

"…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …"

Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency حسب Caiping Zhang (6745976)

"…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …"

Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency حسب Caiping Zhang (6745976)

"…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …"

Data Sheet 2_MiR-216a-5p protects against high glucose-induced HMC injury by targeting the HMGB1/RAGE signaling pathway.pdf حسب Juan Zhang (48597)

"…In vitro experiments, in which high glucose concentrations were used to stimulate human mesangial cells (HMCs), demonstrated a significant increase in the protein level of high mobility group box 1 (HMGB1) and a marked decrease in miR-216a-5p expression (all P < 0.05). …"

Data Sheet 1_MiR-216a-5p protects against high glucose-induced HMC injury by targeting the HMGB1/RAGE signaling pathway.docx حسب Juan Zhang (48597)

"…In vitro experiments, in which high glucose concentrations were used to stimulate human mesangial cells (HMCs), demonstrated a significant increase in the protein level of high mobility group box 1 (HMGB1) and a marked decrease in miR-216a-5p expression (all P < 0.05). …"

Effects of receptor blockers on VO-induced hypertrophy in published experiments and the calibrated model. حسب Johane H. Bracamonte (12883844)

"…<p>a) Simulations of β blockers (βB) at both early and late stages of VO, simulations (boxplots) were equally likely to predict increases or decreases, with no net effect on average. …"

Cytokines produced by CD4+ T cells after <i>in vitro</i> restimulation with rTcj2 protein. حسب Leroy Versteeg (4248760)

HSF2 expression sustains KSHV infection in KLEC. حسب Lorenza Cutrone (21144285)

Primers used in ChIP-PCR. حسب Lorenza Cutrone (21144285)

"…In lymphatic endothelial cells infected with KSHV and in gastric cancer cells positive for EBV, ectopic HSF2 enhances the expression of lytic genes; While knocking down HSF2 significantly decreases their expression. HSF2 overexpression is accompanied by decreased levels of repressive histone marks at the promoters of the lytic regulators KSHV ORF50 and EBV BZLF1, both characterized by poised chromatin features. …"

Primers used in RTqPCR. حسب Lorenza Cutrone (21144285)

"…In lymphatic endothelial cells infected with KSHV and in gastric cancer cells positive for EBV, ectopic HSF2 enhances the expression of lytic genes; While knocking down HSF2 significantly decreases their expression. HSF2 overexpression is accompanied by decreased levels of repressive histone marks at the promoters of the lytic regulators KSHV ORF50 and EBV BZLF1, both characterized by poised chromatin features. …"

HSF2 regulates ORF50 expression in an RTA independent manner. حسب Lorenza Cutrone (21144285)

HSF2 regulates lytic gene expression in latency and during the lytic phase. حسب Lorenza Cutrone (21144285)