Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency by Caiping Zhang (6745976)

“…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …”

Proteome and Metabolome Profiling of Anticoagulant Disorders Induced by Familial Protein S Deficiency by Caiping Zhang (6745976)

“…Furthermore, 9 differential proteins correlated significantly with protein S, comprising A2M, AGT, APOE, FGG, GPLD1, IGHV1–69, CFHR5, CPN2, and CA1. …”

HSF2 expression sustains KSHV infection in KLEC. by Lorenza Cutrone (21144285)

Primers used in ChIP-PCR. by Lorenza Cutrone (21144285)

“…In lymphatic endothelial cells infected with KSHV and in gastric cancer cells positive for EBV, ectopic HSF2 enhances the expression of lytic genes; While knocking down HSF2 significantly decreases their expression. HSF2 overexpression is accompanied by decreased levels of repressive histone marks at the promoters of the lytic regulators KSHV ORF50 and EBV BZLF1, both characterized by poised chromatin features. …”

Primers used in RTqPCR. by Lorenza Cutrone (21144285)

“…In lymphatic endothelial cells infected with KSHV and in gastric cancer cells positive for EBV, ectopic HSF2 enhances the expression of lytic genes; While knocking down HSF2 significantly decreases their expression. HSF2 overexpression is accompanied by decreased levels of repressive histone marks at the promoters of the lytic regulators KSHV ORF50 and EBV BZLF1, both characterized by poised chromatin features. …”

HSF2 regulates ORF50 expression in an RTA independent manner. by Lorenza Cutrone (21144285)

HSF2 regulates lytic gene expression in latency and during the lytic phase. by Lorenza Cutrone (21144285)

Complementary conditioning measures in healthy subjects. by Gabriela Ribeiro (4748373)

“…<b>(I) Novelty</b> ratings significantly decreased from pre to post-conditioning (F_(1,51) = 10.2, <i>P</i> = 0.002; post hoc CS^-, <i>P</i> = 0.0001; post hoc CS⁺, <i>P</i> = 0.01) but similarly for both stimuli (F_{(1, 51)} = 0.17, <i>P</i> = 0.7; Interaction: F_{(1, 51)} = 1.1, <i>P</i> = 0.3). …”

HEK.219 cells were treated with the indicated siRNAs for 24h. by Lorenza Cutrone (21144285)

HSF2 binds the ORF50 promoter and remodels the local chromatin landscape. by Lorenza Cutrone (21144285)

HSF2 is degraded during KSHV lytic reactivation. by Lorenza Cutrone (21144285)

All data points from Fig 2. by Sara Hijazi (21656615)

“…We also found a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implicated in temporal control of PV interneuron firing during cortical network activity. …”

All data points from Fig 5. by Sara Hijazi (21656615)

“…We also found a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implicated in temporal control of PV interneuron firing during cortical network activity. …”

All data points from Fig 8. by Sara Hijazi (21656615)

“…We also found a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implicated in temporal control of PV interneuron firing during cortical network activity. …”

All data points from Fig 3. by Sara Hijazi (21656615)

“…We also found a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implicated in temporal control of PV interneuron firing during cortical network activity. …”

All data points from Fig 1. by Sara Hijazi (21656615)

“…We also found a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implicated in temporal control of PV interneuron firing during cortical network activity. …”

All data points from Fig 4. by Sara Hijazi (21656615)

“…We also found a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implicated in temporal control of PV interneuron firing during cortical network activity. …”

All data points from Fig 9. by Sara Hijazi (21656615)

“…We also found a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implicated in temporal control of PV interneuron firing during cortical network activity. …”

All data points from Fig 7. by Sara Hijazi (21656615)

“…We also found a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implicated in temporal control of PV interneuron firing during cortical network activity. …”

All data points from Fig 6. by Sara Hijazi (21656615)

“…We also found a significant impairment in PV interneuron autaptic self-inhibitory transmission, a feature implicated in temporal control of PV interneuron firing during cortical network activity. …”