Changes in the antioxidant enzyme activities of the wheat cultivars ZM 9023 and YM 15 during artificial aging. by Bang-Bang Li (12329215)

Subjects:

Heat maps of the correlation between seed vigor and carbonyl protein content, reactive oxygen content, non-enzymatic antioxidant contents and antioxidant enzyme activities of wheat... by Bang-Bang Li (12329215)

Subjects:

Changes in the superoxide anion (O2−) and hydrogen peroxide (H₂O₂) contents of the wheat cultivars ZM 9023 and YM 15 during artificial aging. by Bang-Bang Li (12329215)

Subjects:

Changes in the glutathione contents of the wheat cultivars ZM 9023 and YM 15 during artificial aging. by Bang-Bang Li (12329215)

Subjects:

Changes in the carbonyl protein contents of the wheat cultivars ZM 9023 and YM 15 during artificial aging. by Bang-Bang Li (12329215)

Subjects:

Changes in the ultrastructures of wheat embryo cells of the cultivars ZM 9023 and YM 15 during artificial aging. by Bang-Bang Li (12329215)

Subjects:

Effects of the artificial aging treatment on wheat seed vigor. by Bang-Bang Li (12329215)

Subjects:

Results of GO analysis showing significantly enriched biological process (BP), cellular component (CC) and molecular function (MF) in genes with significantly decreased expression... by Lucy Granat (5570813)

Model of the role of increased ROS in chromosome missegregation. by Zachary J. Hough (20840430)

Subjects:

DataSheet2_Aristolochic acid I exposure decreases oocyte quality.xlsx by Weidong Li (292436)

“…Moreover, embryo development potential was also dramatically decreased. Further analyses revealed that AAI exposure significantly decreased mitochondrial membrane potential and ATP synthesis and increased the level of reactive oxygen species (ROS), implying impaired mitochondrial function. …”

DataSheet1_Aristolochic acid I exposure decreases oocyte quality.xlsx by Weidong Li (292436)

“…Moreover, embryo development potential was also dramatically decreased. Further analyses revealed that AAI exposure significantly decreased mitochondrial membrane potential and ATP synthesis and increased the level of reactive oxygen species (ROS), implying impaired mitochondrial function. …”

DataSheet4_Aristolochic acid I exposure decreases oocyte quality.pdf by Weidong Li (292436)

“…Moreover, embryo development potential was also dramatically decreased. Further analyses revealed that AAI exposure significantly decreased mitochondrial membrane potential and ATP synthesis and increased the level of reactive oxygen species (ROS), implying impaired mitochondrial function. …”

DataSheet3_Aristolochic acid I exposure decreases oocyte quality.pdf by Weidong Li (292436)

“…Moreover, embryo development potential was also dramatically decreased. Further analyses revealed that AAI exposure significantly decreased mitochondrial membrane potential and ATP synthesis and increased the level of reactive oxygen species (ROS), implying impaired mitochondrial function. …”

Significantly decreased necrosis observed on wheat leaves inoculated with <i>ΔZtknr4</i>. by Erika Kroll (20537020)

Trend of TI values with age: The TI values decreased significantly with the increase of age, * p<0.01 vs other groups. by Yunmeng Da (707943)

Decrease of admissions and lead diagnoses. by Petr Fulin (8380320)

Subjects:

Copper-Based Complexes with Adamantane Ring-Conjugated <i>bis</i>(3,5-Dimethyl-pyrazol-1-yl)acetate Ligand as Promising Agents for the Treatment of Glioblastoma by Maria Beatrice Morelli (6281642)

Subjects:

GLS inhibition decreases IL-1β secretion <i>in vitro</i>. by Bárbara Ponzilacqua-Silva (17002156)

The alleviating effect of exogenous prohexadione-calcium (Pro-Ca) on the membrane damage and ROS accumulation of rice under salt stress. by Rongjun Zhang (2359339)

An old medicine as a new drug to prevent mitochondrial complex I from producing oxygen radicals by Dominique Detaille (3768979)

“…., as a radical scavenger) but unexpectedly decreases mitochondrial reactive oxygen species (ROS/H₂O₂) production by acting as a specific inhibitor of ROS production at the I_Q site of complex I of the mitochondrial respiratory chain. …”