NS effectively modulates the PI3K/Akt/mTOR signaling pathway and inhibits lipid transport, inflammation, and apoptosis in macrophages by enhancing autophagy. by Yihua Wang (491766)

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NS co-suppresses Ox-LDL-induced apoptosis in RAW264.7 cells. by Yihua Wang (491766)

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NS enhances autophagy inhibits atherosclerosis in RAW264.7 cells. by Yihua Wang (491766)

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NS affects lipid transport in ApoE^-/-mouse models. by Yihua Wang (491766)

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Network pharmacology analysis predicted the targets of NS on atherosclerosis. by Yihua Wang (491766)

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Histopathological analysis of mouse kidney tissue under different treatments. by Yihua Wang (491766)

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NS restores macrophage autophagy and alleviates the progression of atherosclerosis by inhibiting the PI3K/Akt/mTOR pathway. by Yihua Wang (491766)

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NS suppresses inflammation induced by Ox-LDL in RAW264.7 cells. by Yihua Wang (491766)

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Significant associations in GWAS. by Dai Nogimura (21445649)

“…<div><p>Decreased nitric oxide (NO) production from the vascular endothelium is a major factor for vascular aging. …”

Conditional ablation of <i>Ybx1</i> leads to a reduction in cortical progenitor cell pool and a decrease in cortical thickness. by Jian Zhang (1682)

Demonstration of decreased Aβ accumulation by DA-9601 treatment. by Mijung Lee (397790)

Decreased clonogenic capacity of U87MG and U251MG GB cells following treatment with SHG-44 and cis-platin at equal concentrations. by Denis Mustafov (19137870)

Table 3_Ferrostatin-1 improves acute sepsis-induced cardiomyopathy via inhibiting neutrophil infiltration through impaired chemokine axis.xlsx by Jialin Li (382950)

“…Consistently, expression of neutrophil associated chemokines (Ccrl2, Cxcl2, Cxcl3 and Cxcl5) as well as extracellular matrix (ECM) degradation enzymes (Adamts1, Adamts4, Adamts9 and Mmp8) significantly decreased in Fer-1 pre-treated CLP heart.…”

Table 1_Ferrostatin-1 improves acute sepsis-induced cardiomyopathy via inhibiting neutrophil infiltration through impaired chemokine axis.xlsx by Jialin Li (382950)

“…Consistently, expression of neutrophil associated chemokines (Ccrl2, Cxcl2, Cxcl3 and Cxcl5) as well as extracellular matrix (ECM) degradation enzymes (Adamts1, Adamts4, Adamts9 and Mmp8) significantly decreased in Fer-1 pre-treated CLP heart.…”

Table 2_Ferrostatin-1 improves acute sepsis-induced cardiomyopathy via inhibiting neutrophil infiltration through impaired chemokine axis.xlsx by Jialin Li (382950)

“…Consistently, expression of neutrophil associated chemokines (Ccrl2, Cxcl2, Cxcl3 and Cxcl5) as well as extracellular matrix (ECM) degradation enzymes (Adamts1, Adamts4, Adamts9 and Mmp8) significantly decreased in Fer-1 pre-treated CLP heart.…”

Image 1_Ferrostatin-1 improves acute sepsis-induced cardiomyopathy via inhibiting neutrophil infiltration through impaired chemokine axis.jpeg by Jialin Li (382950)

“…Consistently, expression of neutrophil associated chemokines (Ccrl2, Cxcl2, Cxcl3 and Cxcl5) as well as extracellular matrix (ECM) degradation enzymes (Adamts1, Adamts4, Adamts9 and Mmp8) significantly decreased in Fer-1 pre-treated CLP heart.…”

Image 2_Ferrostatin-1 improves acute sepsis-induced cardiomyopathy via inhibiting neutrophil infiltration through impaired chemokine axis.jpeg by Jialin Li (382950)

“…Consistently, expression of neutrophil associated chemokines (Ccrl2, Cxcl2, Cxcl3 and Cxcl5) as well as extracellular matrix (ECM) degradation enzymes (Adamts1, Adamts4, Adamts9 and Mmp8) significantly decreased in Fer-1 pre-treated CLP heart.…”

Schematic diagram of the molecular mechanism by which TRIM37 improves MS by stabilizing PEX5 through monoubiquitination. by Lai Jiang (2401513)

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BT9 did not improve survival in the orthotopic xenograft model of D425 medulloblastoma. by Zahra Motahari (6814013)

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PEDV and PDCoV infection impairs the cilium. by Tenghan Zhuang (22238809)