Search alternatives:
significantly modulated » significantly mediated (Expand Search), significantly correlated (Expand Search), significantly related (Expand Search)
gap decrease » gain decreased (Expand Search), mean decrease (Expand Search), step decrease (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
significantly modulated » significantly mediated (Expand Search), significantly correlated (Expand Search), significantly related (Expand Search)
gap decrease » gain decreased (Expand Search), mean decrease (Expand Search), step decrease (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
-
1
-
2
-
3
-
4
-
5
Syndecan ectodomain fragments decrease endothelial resistance via rho kinase.
Published 2019“…In the presence of RWJ56110, rhS3ED fragments mediated a significant decrease in TER response (n = 3, p<0.0001). …”
-
6
-
7
-
8
-
9
-
10
-
11
Enrichment analysis of genes from the significant MS module.
Published 2025“…<p><b>(a)</b> Venn diagram showing the intersection of DEGs and significant module genes from WGCNA, resulting in 46 key intersection genes. …”
-
12
-
13
-
14
-
15
-
16
-
17
BMAA decreases cell proliferation.
Published 2023“…<p>Treatment of IMR-32 cells with increasing doses of BMAA over a 24 hour period resulted in decreased cell number and inhibition of cell proliferation as determined by microscopy and quantified by XTT assay (A and B). …”
-
18
Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket
Published 2021“…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
-
19
Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket
Published 2021“…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
-
20