بدائل البحث:
selected diseases » neglected disease (توسيع البحث), detected disease (توسيع البحث), related disease (توسيع البحث)
four selected » five selected (توسيع البحث)
a decrease » _ decreased (توسيع البحث), _ decreases (توسيع البحث)
selected diseases » neglected disease (توسيع البحث), detected disease (توسيع البحث), related disease (توسيع البحث)
four selected » five selected (توسيع البحث)
a decrease » _ decreased (توسيع البحث), _ decreases (توسيع البحث)
-
1
-
2
-
3
-
4
HFD induces LDs and decreases ER and mitochondria in nephrocytes.
منشور في 2021"…(<b>E</b>) Low and high magnification views of pericardial nephrocytes (dotted outlines) from STD and HFD larvae, showing that HFD decreases mitochondria (marked with anti-ATP5A) and ER (marked with anti-KDEL) but increases LDs (marked with LipidTOX). …"
-
5
-
6
-
7
-
8
-
9
-
10
-
11
-
12
-
13
-
14
Table_1_Human Complement C4B Allotypes and Deficiencies in Selected Cases With Autoimmune Diseases.docx
منشور في 2021"…Second, in healthy White subject MS630, a C-nucleotide deletion at codon-755 led to frameshift mutations in his single C4B gene, which was a private mutation. Third, in European family E94 with multiplex lupus-related mortality and low serum C4 levels, the culprit was a recurrent haplotype with HLA-A30, B18 and DR7 that segregated with two defective C4B genes and identical mutations at the donor splice site of intron-28. …"
-
15
PSG-specific <i>Fzr</i> mutation decreases the expression of silk protein genes.
منشور في 2023"…<p>(A) <i>Fzr</i> mutation in the PSG resulted in thinner cocoon compared with controls. …"
-
16
-
17
-
18
DataSheet_1_Human Complement C4B Allotypes and Deficiencies in Selected Cases With Autoimmune Diseases.docx
منشور في 2021"…Second, in healthy White subject MS630, a C-nucleotide deletion at codon-755 led to frameshift mutations in his single C4B gene, which was a private mutation. Third, in European family E94 with multiplex lupus-related mortality and low serum C4 levels, the culprit was a recurrent haplotype with HLA-A30, B18 and DR7 that segregated with two defective C4B genes and identical mutations at the donor splice site of intron-28. …"
-
19
-
20