يعرض 1 - 20 نتائج من 87,920 نتيجة بحث عن '(( third ((((_ decrease) OR (a decrease))) OR (related diseases)) ) OR ( four selected diseases ))', وقت الاستعلام: 1.79s تنقيح النتائج
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    Third. حسب Cuicui Liu (1496260)

    منشور في 2024
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    HFD induces LDs and decreases ER and mitochondria in nephrocytes. حسب Aleksandra Lubojemska (10746241)

    منشور في 2021
    "…(<b>E</b>) Low and high magnification views of pericardial nephrocytes (dotted outlines) from STD and HFD larvae, showing that HFD decreases mitochondria (marked with anti-ATP5A) and ER (marked with anti-KDEL) but increases LDs (marked with LipidTOX). …"
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    Table_1_Human Complement C4B Allotypes and Deficiencies in Selected Cases With Autoimmune Diseases.docx حسب Danlei Zhou (6669173)

    منشور في 2021
    "…Second, in healthy White subject MS630, a C-nucleotide deletion at codon-755 led to frameshift mutations in his single C4B gene, which was a private mutation. Third, in European family E94 with multiplex lupus-related mortality and low serum C4 levels, the culprit was a recurrent haplotype with HLA-A30, B18 and DR7 that segregated with two defective C4B genes and identical mutations at the donor splice site of intron-28. …"
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    PSG-specific <i>Fzr</i> mutation decreases the expression of silk protein genes. حسب Wenliang Qian (639395)

    منشور في 2023
    "…<p>(A) <i>Fzr</i> mutation in the PSG resulted in thinner cocoon compared with controls. …"
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    Flow diagram of study selection. حسب Amani Kacem (20521536)

    منشور في 2025
    الموضوعات:
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    Case selection flow chart. حسب Sara Garcia-Ptacek (693846)

    منشور في 2023
    الموضوعات:
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    DataSheet_1_Human Complement C4B Allotypes and Deficiencies in Selected Cases With Autoimmune Diseases.docx حسب Danlei Zhou (6669173)

    منشور في 2021
    "…Second, in healthy White subject MS630, a C-nucleotide deletion at codon-755 led to frameshift mutations in his single C4B gene, which was a private mutation. Third, in European family E94 with multiplex lupus-related mortality and low serum C4 levels, the culprit was a recurrent haplotype with HLA-A30, B18 and DR7 that segregated with two defective C4B genes and identical mutations at the donor splice site of intron-28. …"
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