Showing 1 - 20 results of 21,841 for search '(( via ((((a decrease) OR (_ decrease))) OR (larger decrease)) ) OR ( _ part decrease ))', query time: 0.65s Refine Results
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    DataSheet3_Coprophagy Prevention Decreases the Reproductive Performance and Granulosa Cell Apoptosis via Regulation of CTSB Gene in Rabbits.docx by Guohua Song (5152595)

    Published 2022
    “…Overexpression of CTSB increased secretion of progesterone and estradiol, partly via upregulation of CYP19A1 while inhibition of CTSB decreased progesterone secretion partly via downregulation of the StAR gene. …”
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    DataSheet2_Coprophagy Prevention Decreases the Reproductive Performance and Granulosa Cell Apoptosis via Regulation of CTSB Gene in Rabbits.ZIP by Guohua Song (5152595)

    Published 2022
    “…Overexpression of CTSB increased secretion of progesterone and estradiol, partly via upregulation of CYP19A1 while inhibition of CTSB decreased progesterone secretion partly via downregulation of the StAR gene. …”
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    DataSheet1_Coprophagy Prevention Decreases the Reproductive Performance and Granulosa Cell Apoptosis via Regulation of CTSB Gene in Rabbits.ZIP by Guohua Song (5152595)

    Published 2022
    “…Overexpression of CTSB increased secretion of progesterone and estradiol, partly via upregulation of CYP19A1 while inhibition of CTSB decreased progesterone secretion partly via downregulation of the StAR gene. …”
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    Decrease in apoptosis in the ischemic myocardium. by Laura A. Scrimgeour (9367459)

    Published 2020
    “…<p>There is an increase seen in JNK, pBAD, and pBAD/BAD with a decrease in pBCL-2, pBCL-2/BCL-2, and Caspase 3. …”
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    Data_Sheet_1_A de novo Mutation in the MTUS1 Gene Decreases the Risk of Non-compaction of Ventricular Myocardium via the Rac1/Cdc42 Pathway.pdf by Xuehan Bai (6901679)

    Published 2019
    “…The phosphorylation of Rac1/Cdc42 in the mutation group was significantly lower than in the wild type group.</p><p>Conclusions: A de novo mutation in MTUS1 decreased the stability of microtubules and increased cell polarity via the Rac1/Cdc42 pathway, which may partly elucidate the mechanism underlying cellular protection in NVM.…”