Showing 141 - 160 results of 47,702 for search '(( via ((((greater decrease) OR (a decrease))) OR (larger decrease)) ) OR ( i large increases ))', query time: 1.41s Refine Results
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    Image_1_Mice with lung airway ciliopathy develop persistent Mycobacterium abscessus lung infection and have a proinflammatory lung phenotype associated with decreased T regulatory... by Audrey Nava (14173695)

    Published 2022
    “…T cells subset analysis showed a decrease in the percentage of CD4<sup>+</sup>FoxP3<sup>+</sup> T cells in the total lymphocyte population in the lungs of IFT88 KO mice relative to IFT88 control mice. …”
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    Table1_Sevoflurane Increases Hippocampal Theta Oscillations and Impairs Memory Via TASK-3 Channels.DOCX by Linlin Han (3709822)

    Published 2021
    “…<p>Sevoflurane can induce memory impairment during clinical anesthesia; however, the underlying mechanisms are largely unknown. TASK-3 channels are one of the potential targets of sevoflurane. …”
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    Table_1_Prenatal exposure to inflammation increases anxiety-like behaviors in F1 and F2 generations: possible links to decreased FABP7 in hippocampus.DOCX by Jing Chen (4762)

    Published 2022
    “…Overall, gestational LPS exposure in the F0 generation increased anxiety levels and decreased FABP7 expression levels in the F1 generation, which carried over to the F2 generation, and the intergenerational effects were mainly transferred via the maternal lineage. …”
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    Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket by Lilia Leisle (11356934)

    Published 2021
    “…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
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    Dynorphin Neuropeptides Decrease Apparent Proton Affinity of ASIC1a by Occluding the Acidic Pocket by Lilia Leisle (11356934)

    Published 2021
    “…Prolonged acidosis, as it occurs during ischemic stroke, induces neuronal death via acid-sensing ion channel 1a (ASIC1a). Concomitantly, it desensitizes ASIC1a, highlighting the pathophysiological significance of modulators of ASIC1a acid sensitivity. …”
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