Showing 5,901 - 5,920 results of 27,058 for search '(( via ((a decrease) OR (linear decrease)) ) OR ( a ((largest decrease) OR (larger decrease)) ))', query time: 0.78s Refine Results
  1. 5901

    Cantharidin Induced Oral Squamous Cell Carcinoma Cell Apoptosis via the JNK-Regulated Mitochondria and Endoplasmic Reticulum Stress-Related Signaling Pathways by Chin-Chuan Su (279690)

    Published 2016
    “…<div><p>Oral cancer is a subtype of head and neck cancer which represents 2.65% of all human malignancies. …”
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  11. 5911

    MHY446 induces apoptosis via reactive oxygen species-mediated endoplasmic reticulum stress in HCT116 human colorectal cancer cells by Yu Ra Ahn (17592166)

    Published 2023
    “…<p>This study investigated the potential of a newly synthesized histone deacetylase (HDAC) inhibitor, MHY446, in inducing cell death in HCT116 colorectal cancer cells and compared its activity with that of suberoylanilide hydroxamic acid (SAHA), a well-known HDAC inhibitor. …”
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    DataSheet1_Effects of Abelmoschus manihot (L.) and its combination with irbesartan in the treatment of diabetic nephropathy via the gut–kidney axis.PDF by Hongmei Yu (584700)

    Published 2024
    “…Furthermore, the S100a8, S100a9, Trem1, and Mmp7 genes in the kidneys were downregulated. …”
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    Supplementary Material for: Topical Vitamin D Receptor Antagonist/Partial-Agonist Treatment Induces Epidermal Hyperproliferation via RARγ Signaling Pathways by Lucas R. (9321548)

    Published 2020
    “…In summary, we conclude that epidermal hyperproliferation of mouse skin in response to a topically administered vitamin D receptor antagonist/partial agonist (ZK159222) is induced via increased retinoic acid synthesis, retinoic acid levels and increased RARγ-mediated pathways.…”
  19. 5919

    GLI3 Repressor Controls Nephron Number via Regulation of <em>Wnt11</em> and <em>Ret</em> in Ureteric Tip Cells by Jason E. Cain (258860)

    Published 2009
    “…HH-dependent signaling increases levels of GLI transcriptional activators and decreases processing of GLI3 to a shorter transcriptional repressor. …”
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