Image_2_Proteasome-Mediated Regulation of Cdhr1a by Siah1 Modulates Photoreceptor Development and Survival in Zebrafish.TIF by Warlen Pereira Piedade (9671738)

“…Taken together, our work provides the first evidence that Cdhr1a plays a role during early photoreceptor development and that Cdhr1a is regulated by Siah1 via the UPS.…”

Image_4_Proteasome-Mediated Regulation of Cdhr1a by Siah1 Modulates Photoreceptor Development and Survival in Zebrafish.TIF by Warlen Pereira Piedade (9671738)

“…Taken together, our work provides the first evidence that Cdhr1a plays a role during early photoreceptor development and that Cdhr1a is regulated by Siah1 via the UPS.…”

Image_1_Proteasome-Mediated Regulation of Cdhr1a by Siah1 Modulates Photoreceptor Development and Survival in Zebrafish.TIF by Warlen Pereira Piedade (9671738)

“…Taken together, our work provides the first evidence that Cdhr1a plays a role during early photoreceptor development and that Cdhr1a is regulated by Siah1 via the UPS.…”

Image_3_Proteasome-Mediated Regulation of Cdhr1a by Siah1 Modulates Photoreceptor Development and Survival in Zebrafish.TIF by Warlen Pereira Piedade (9671738)

“…Taken together, our work provides the first evidence that Cdhr1a plays a role during early photoreceptor development and that Cdhr1a is regulated by Siah1 via the UPS.…”

Study timeline and methodological overview. by Robert Botelho (20308297)

Table 1_The impact of environmental and health messaging, pulse prevalence, and filtering tools on pulse-based food choice and nutrient profiles: a randomized online experiment.doc... by Christopher R. Gustafson (6077870)

“…</p>Discussion<p>Combining messaging with filtering resulted in larger relative increases in pulse choices, suggesting that decreasing product search and identification costs may be an important component of interventions. …”

Video_2_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_5_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_4_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_3_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_6_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Video_1_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.mp4 by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Image_1_The TRPV4 Agonist GSK1016790A Regulates the Membrane Expression of TRPV4 Channels.pdf by Sara Baratchi (357850)

“…Further, we show that the density of TRPV4 at the plasma membrane decreased within 20 min, as they translocate to the recycling endosomes and that the surface density is dependent on the release of calcium from the intracellular stores and is controlled via a PI3K, PKC, and RhoA signaling pathway.…”

Schematic representation of the possible role for NAT1 in the regulation of ROS production and apoptosis. by LiLi Wang (1582915)

“…This model suggests NAT1 may be a target to decrease intracellular GOF p53.</p>…”

Data Sheet 1_Global, regional, and national burden of pneumococcal disease among children and adolescents aged <20 years from 1990 to 2021: a predictive analysis.docx by Gan-min Wang (21188933)

“…Despite a global decrease in burden, significant regional and socioeconomic disparities, as well as long-term trends, are not fully understood.…”

PE_HP during Graded Head-up Tilt. by Alberto Porta (223657)

The effect of repeated mechanical stimulations. by Ulisse Bocchero (9128127)

Luciferase expression in mice after intravenous injection rH17d’-Ad5 vectors as revealed by live BLI. by Christopher M. Gentile (6640331)

Primers used for RT-PCR. by Quankuan Gu (20539301)

“…</p><p>Results</p><p>BLM caused a large decrease in body weight in mice. However, the intraperitoneal injection of SS-31 slowed down the body weight loss in the mice. …”

DataSheet2_WNT5A Interacts With FZD5 and LRP5 to Regulate Proliferation and Self-Renewal of Endometrial Mesenchymal Stem-Like Cells.docx by Tianqi Li (1335987)

“…Myometrial cells act as a component of the niche and regulate the stem cell fate through the activation of WNT/β-catenin signaling via WNT5A. …”