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largest decrease » largest decreases (Expand Search), marked decrease (Expand Search)
linear decrease » linear increase (Expand Search)
larger decrease » marked decrease (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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Characterization of passage 1 (P1) bone marrow-derived canine mesenchymal stromal cells (cMSCs).
Published 2022Subjects: -
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Image_8_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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1129
Image_7_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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1130
Image_4_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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Image_6_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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Image_5_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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Image_10_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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Image_1_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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Image_2_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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Image_3_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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Image_9_Orexin-A Exerts Neuroprotective Effects via OX1R in Parkinson’s Disease.TIF
Published 2018“…The upregulation of BDNF is mainly concerned with PI3K and PKC signaling pathways via OX1R. The present study demonstrated that orexin-A exerted neuroprotective effects on MPTP parkinsonian mice, which may imply orexin-A as a potential therapeutic target for PD.…”
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The G Protein-Coupled Receptor RAI3 Is an Independent Prognostic Factor for Pancreatic Cancer Survival and Regulates Proliferation via STAT3 Phosphorylation
Published 2017“…Especially the loss of apical RAI3 expression represents an independent prognostic parameter for overall survival of patients with pancreatic cancer. Suppression of GPRC5a results in decreased cell growth, proliferation and migration in pancreatic cancer cell lines via a STAT3 modulated pathway, independent from ERK activation.…”
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