Quantitative dynamics of <i>Salmonella</i> and <i>E</i>. <i>coli</i> in feces of feedlot cattle treated with ceftiofur and chlortetracycline حسب Naomi Ohta (735090)

"…The purpose of this analysis was to quantify the dynamics of <i>Salmonella</i> using colony counting (via a spiral-plating method) and hydrolysis probe-based qPCR (TaqMan^® qPCR). …"

Perturbing gene expression via mitotic recombination. حسب Sebastian M. Bernasek (8525829)

Multi-Clinic Quality Improvement Initiative Increases Continuous Glucose Monitoring Use Among Adolescents and Young Adults With Type 1 Diabetes حسب Priya Prahalad (50952)

"…This article reports on a quality improvement (QI) initiative of the T1D Exchange Quality Improvement Collaborative to increase CGM use among patients in this age-group. …"

Primers used in this study. حسب Hadi Alizadeh (20854757)

DataSheet3_Zebrafish cobll1a regulates lipid homeostasis via the RA signaling pathway.ZIP حسب Ting Zeng (1521649)

"…</p>Methods:<p>In this study, we established cobll1a mutant lines using the CRISPR/Cas9-mediated gene knockout technique. …"

DataSheet4_Zebrafish cobll1a regulates lipid homeostasis via the RA signaling pathway.ZIP حسب Ting Zeng (1521649)

"…</p>Methods:<p>In this study, we established cobll1a mutant lines using the CRISPR/Cas9-mediated gene knockout technique. …"

DataSheet5_Zebrafish cobll1a regulates lipid homeostasis via the RA signaling pathway.docx حسب Ting Zeng (1521649)

"…</p>Methods:<p>In this study, we established cobll1a mutant lines using the CRISPR/Cas9-mediated gene knockout technique. …"

DataSheet1_Zebrafish cobll1a regulates lipid homeostasis via the RA signaling pathway.ZIP حسب Ting Zeng (1521649)

"…</p>Methods:<p>In this study, we established cobll1a mutant lines using the CRISPR/Cas9-mediated gene knockout technique. …"

DataSheet2_Zebrafish cobll1a regulates lipid homeostasis via the RA signaling pathway.ZIP حسب Ting Zeng (1521649)

"…</p>Methods:<p>In this study, we established cobll1a mutant lines using the CRISPR/Cas9-mediated gene knockout technique. …"

Image_5_TRAPS mutations in Tnfrsf1a decrease the responsiveness to TNFα via reduced cell surface expression of TNFR1.tif حسب Takahiko Akagi (13138338)

"…<p>Tumor necrosis factor (TNF) receptor-associated periodic syndrome (TRAPS) is an autoinflammatory periodic fever syndrome associated with heterozygous mutations in TNFRSF1A, which encodes TNF receptor type I (TNFR1). Although possible proinflammatory mechanisms have been proposed, most previous studies were performed using in vitro overexpression models, which could lead to undesirable inflammatory responses due to artificial overexpression. …"

Image_1_TRAPS mutations in Tnfrsf1a decrease the responsiveness to TNFα via reduced cell surface expression of TNFR1.tif حسب Takahiko Akagi (13138338)

"…<p>Tumor necrosis factor (TNF) receptor-associated periodic syndrome (TRAPS) is an autoinflammatory periodic fever syndrome associated with heterozygous mutations in TNFRSF1A, which encodes TNF receptor type I (TNFR1). Although possible proinflammatory mechanisms have been proposed, most previous studies were performed using in vitro overexpression models, which could lead to undesirable inflammatory responses due to artificial overexpression. …"

Image_4_TRAPS mutations in Tnfrsf1a decrease the responsiveness to TNFα via reduced cell surface expression of TNFR1.tif حسب Takahiko Akagi (13138338)

"…<p>Tumor necrosis factor (TNF) receptor-associated periodic syndrome (TRAPS) is an autoinflammatory periodic fever syndrome associated with heterozygous mutations in TNFRSF1A, which encodes TNF receptor type I (TNFR1). Although possible proinflammatory mechanisms have been proposed, most previous studies were performed using in vitro overexpression models, which could lead to undesirable inflammatory responses due to artificial overexpression. …"

Image_7_TRAPS mutations in Tnfrsf1a decrease the responsiveness to TNFα via reduced cell surface expression of TNFR1.tif حسب Takahiko Akagi (13138338)

"…<p>Tumor necrosis factor (TNF) receptor-associated periodic syndrome (TRAPS) is an autoinflammatory periodic fever syndrome associated with heterozygous mutations in TNFRSF1A, which encodes TNF receptor type I (TNFR1). Although possible proinflammatory mechanisms have been proposed, most previous studies were performed using in vitro overexpression models, which could lead to undesirable inflammatory responses due to artificial overexpression. …"

Image_3_TRAPS mutations in Tnfrsf1a decrease the responsiveness to TNFα via reduced cell surface expression of TNFR1.tif حسب Takahiko Akagi (13138338)

"…<p>Tumor necrosis factor (TNF) receptor-associated periodic syndrome (TRAPS) is an autoinflammatory periodic fever syndrome associated with heterozygous mutations in TNFRSF1A, which encodes TNF receptor type I (TNFR1). Although possible proinflammatory mechanisms have been proposed, most previous studies were performed using in vitro overexpression models, which could lead to undesirable inflammatory responses due to artificial overexpression. …"

Image_6_TRAPS mutations in Tnfrsf1a decrease the responsiveness to TNFα via reduced cell surface expression of TNFR1.tif حسب Takahiko Akagi (13138338)

"…<p>Tumor necrosis factor (TNF) receptor-associated periodic syndrome (TRAPS) is an autoinflammatory periodic fever syndrome associated with heterozygous mutations in TNFRSF1A, which encodes TNF receptor type I (TNFR1). Although possible proinflammatory mechanisms have been proposed, most previous studies were performed using in vitro overexpression models, which could lead to undesirable inflammatory responses due to artificial overexpression. …"

Image_2_TRAPS mutations in Tnfrsf1a decrease the responsiveness to TNFα via reduced cell surface expression of TNFR1.tif حسب Takahiko Akagi (13138338)

"…<p>Tumor necrosis factor (TNF) receptor-associated periodic syndrome (TRAPS) is an autoinflammatory periodic fever syndrome associated with heterozygous mutations in TNFRSF1A, which encodes TNF receptor type I (TNFR1). Although possible proinflammatory mechanisms have been proposed, most previous studies were performed using in vitro overexpression models, which could lead to undesirable inflammatory responses due to artificial overexpression. …"

Brief Report: Increased technology use associated with lower A1C in a large pediatric clinical population حسب G. Todd Alonso (9568724)

"…</p> <p><strong>Results</strong><br> CGM use increased from 32.9% to 75.3%; HCL increased from 0.3% to 27.9%. …"

Active Diffusion of Self-Propelled Particles in Flexible Polymer Networks حسب Yeongjin Kim (10878837)

الموضوعات:

Active Diffusion of Self-Propelled Particles in Flexible Polymer Networks حسب Yeongjin Kim (10878837)

الموضوعات:

Active Diffusion of Self-Propelled Particles in Flexible Polymer Networks حسب Yeongjin Kim (10878837)

الموضوعات: